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GP6 — IL2

Text-mined interactions from Literome

Gollapudi et al., Genes Immun 2000 : P-glycoprotein ( encoded by multidrug resistance genes ) is not required for interleukin-2 secretion in mice and humans
Gorse et al., AIDS Res Hum Retroviruses 2001 (HIV Infections) : There was a possible association between HIV-1 envelope glycoprotein stimulated interleukin 2 secretion and CD8 ( + ) CTLs against HIV-1 envelope glycoprotein, and an inverse relation between lymphocyte proliferation and CTLs against HIV-1 Gag antigens
Kierszenbaum et al., Mol Biochem Parasitol 2002 : Like living Trypanosoma cruzi, its AGC10 membrane glycoprotein inhibits interleukin-2 (IL-2) secretion and membrane expression of CD25, CD122, and CD132 ( the components of the high-affinity IL-2 receptor ) by activated human lymphocytes
Sundar et al., Proc Natl Acad Sci U S A 1991 : Human immunodeficiency virus glycoprotein ( gp120 ) infused into rat brain induces interleukin 1 to elevate pituitary-adrenal activity and decrease peripheral cellular immune responses
Becker et al., Virus Genes 2006 (Hypersensitivity...) : The review of the molecular research on the role of the viral fusion ( F ) and attachment ( G ) glycoproteins of RSV provided information on their role in the virus infection : early in infection the F glycoprotein induces Th1 cells to release the Th1 cytokines IL-2 , IL-12 and IFN-gamma to activate precursors CTLs ( pCTLs ) to become anti-RSV CTLs
Tsujimura et al., J Biol Chem 2006 : We found previously that interleukin-2 , a lymphocyte-activation cytokine, induces P-glycoprotein expression on lymphocytes
Espinoza-Delgado et al., J Immunol 1990 : These data demonstrate that IL-2 enhances c-fms mRNA and c-fms glycoprotein expression suggesting that IL-2, by augmenting expression of macrophage CSF-1 receptors, can lead to prolongation of monocyte mediated tumoricidal activity obtained in the presence of exogenous macrophage CSF-1
Petrarca et al., Microb Pathog 1988 (Virus Diseases) : The purified viral membrane glycoprotein , G protein, and Gs ( secreted G protein that lacks the hydrophobic and intracytoplasmic domains ) both stimulated IL-2 production when added to cultures of A20 and the hybridomas
Kojima et al., J Pharmacobiodyn 1986 (Neoplasms, Experimental) : Furthermore, this serum glycoprotein was found to enhance glucose consumption and interleukin-1 production and cytotoxic activities of mouse peritoneal macrophages
Geiger et al., J Biol Chem 1988 (Liver Neoplasms, Experimental) : Induction of alpha 1-acid glycoprotein by recombinant human interleukin-1 in rat hepatoma cells
Lanier et al., J Exp Med 1988 : Appearance of this glycoprotein on NK cells is IL-2 dependent and closely parallels IL-2 induced cytotoxicity against NK-resistant solid tumor cell targets
Oth et al., Cell Immunol 1987 : The association of the rabies glycoprotein with liposome ( immunosome ) induces an in vitro specific release of interleukin 2
Macfarlan et al., J Immunol 1984 : The intact viral glycoprotein and synthetic peptide R21 stimulated T lymphocytes with surface characteristics of helper cells, and induced the production of interleukin 2 by these lymphocytes
Weston et al., Transpl Immunol 1995 (Kidney Failure, Chronic) : E-Selectin is a 115-kDa cell surface glycoprotein transiently expressed on vascular endothelium in response to interleukin-1 and tumour necrosis factor-alpha with a peak in expression at four hours
Fournier et al., Hepatology 1994 : These results show that ( a ) phenobarbital acts directly on hepatocytes by increasing alpha 1-acid glycoprotein gene expression and ( b ) this effect is mediated by a specific mechanism independent of pathways involved in alpha 1-acid glycoprotein induction by interleukin-1 , interleukin-6 and glucocorticoids
Herbert et al., FEBS Lett 1993 : Endotoxin ( LPS ), interleukin-1 beta (IL-1) and tumor necrosis factor-alpha (TNF) increased the expression of tissue factor, a membrane anchored glycoprotein that initiates blood coagulation on the surface of cultured bovine aortic endothelial cells ( ABAE ) and human monocytes
Andrejko et al., Crit Care Med 1996 (Sepsis) : To test the hypothesis that after cecal ligation and puncture in the rat, there is increased expression of the tumor necrosis factor (TNF)/interleukin-1 dependent , acute-phase reactant alpha 1-acid glycoprotein in the liver, and that this change correlates temporally with increased abundance of TNF-alpha in the hepatic parenchyma but not with circulating concentrations of TNF-alpha