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FOS — KCNH4
Text-mined interactions from Literome
Bruning et al., Proc Natl Acad Sci U S A 2000
:
Thus Rsk-2 is required for growth factor stimulated expression of
c-Fos and transcriptional
activation of
Elk-1 and the serum response factor, but not for activation of CREB or the mitogen activated protein kinase pathway in response to PDGF and IGF-1 stimulation
Wiren et al., J Mol Endocrinol 2004
(MAP Kinase Signaling System) :
Analysis of phosphorylation cascades demonstrated that DHT treatment inhibited phosphoERK1/2 levels, MAP kinase
activation of
Elk-1 , Elk-1 protein and phosphoElk-1 levels, and downstream
AP-1/luciferase reporter activity
Yan et al., Braz J Med Biol Res 2006
:
However, Western blots showed that the induction of
Elk-1 regulated expression of endogenous
c-Fos was not affected by Pim-1
Müller et al., J Cell Biochem 2010
(Insulinoma) :
Expression of dominant negative mutants of c-Jun and
Elk-1 reduced
AP-1 transcriptional activity in INS-1 cells indicating that c-Jun and ternary complex factors are involved in the regulation of AP-1 activity in glucose stimulated insulinoma cells
Thiel et al., Endocrinology 2012
:
The expression of a dominant negative mutant of
Elk-1 reduced c-Fos expression and
prevented the up-regulation of
AP-1 activity as a result of calcium sensing receptor stimulation, indicating that ternary complex factors control both Egr-1- and AP-1 regulated transcription