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FGF1 — IL8
Text-mined interactions from Literome
Thomas et al., Arthritis Rheum 2000
:
Tumor necrosis factor alpha,
interleukin-6 , and epidermal growth factor did not
increase aFGF expression in vitro ; in contrast, transforming growth factor beta1 ( TGFbeta1 ) was found to markedly increase aFGF production by cultured synovial cells
Sandell et al., Osteoarthritis Cartilage 2008
(Arthritis) :
In the presence of IL-1beta,
FGF-18 increased expression of ADAMTS-4, aggrecan, BMP-2, COL2A1, CCL3, CCL4, CCL20, CXCL1, CXCL3, CXCL6, IL-1beta, IL-6, and
IL-8 and decreased ADAMTS-5, MMP-13, CCL2, and CCL8
Nakayama et al., J Oral Sci 2012
:
In human osteoblast-like Saos2 cells,
FGF2 increased the expression of the osteopontin ( SPP1, 16.7-fold ),
interleukin-8 ( IL8, 6.4-fold ) and IL11 ( 4.8-fold ) genes
Chandrasekhar et al., J Cell Physiol 1989
:
Induction of
interleukin-1 receptors on chondrocytes by
fibroblast growth factor : a possible mechanism for modulation of interleukin-1 activity
Iwamoto et al., Immunol Lett 1989
:
Furthermore,
interleukin 1 (IL-1) suppressed by 50-60 % the incorporation of [ 3H ] thymidine into DNA in high density chondrocyte cultures on plastic dishes in the
presence of 10 % serum or
fibroblast growth factor , although it increased twofold this incorporation in 0.3 % serum alone
Norioka et al., Jpn J Cancer Res 1994
(Neovascularization, Pathologic) :
Interaction of
interleukin-1 and interferon-gamma on
fibroblast growth factor induced angiogenesis
Lingen et al., Lab Invest 1996
(Carcinoma, Squamous Cell...) :
Clinically achievable doses of RA rapidly caused large- and small-vessel endothelial cells to become refractory to stimulation of migration either by tumor conditioned media or purified angiogenic factors (
a-fibroblast growth factor (aFGF) , bFGF, vascular endothelial GF, platelet
derived GF, TGF beta-1, and
IL-8 )