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CALM3 — IRF6

Text-mined interactions from Literome

Blease et al., J Immunol 1999 (Lung Diseases...) : S. aureus and S. pyogenes LTA ( 30 microg/ml ) abolished LPS induced CAM expression, and S. aureus LTA reduced LPS induced IL-8 release
Shinkai et al., Am J Physiol Lung Cell Mol Physiol 2006 : In the presence of LPS , both CAM and AZM dose-dependently increased IL-8 secretion over 24 h, but after 5 days of exposure to 10 microg/ml CAM there is suppression of IL-8 ( P < 0.001 )
Mishra et al., J Biol Chem 2005 (Calcium Signaling) : Taken together, these results suggest that LPS- and TNF-alpha induced c-IAP2 expression and its associated antiapoptotic survival signals in THP-1 cells are regulated selectively by CaM/CaMKII through NF-kappaB activation
Smallwood et al., Biochemistry 2006 : To understand regulatory mechanisms underlying macrophage activation, we have investigated changes in the abundance of calmodulin (CaM) and iNOS in response to the bacterial cell wall component lipopolysaccharide (LPS) using RAW 264.7 macrophages
Nicolete et al., Phytomedicine 2009 : In vitro, the effects of budlein A on LPS induced cytokine, chemokine and nitrites release, T-cell proliferative response as well as cell adhesion molecule expression (CAM) were evaluated
Zuckerman et al., Thromb Res 1988 : As many of these mediators activate protein kinase C, the effect of calmodulin and protein kinase C inhibitors on IL-1, TNF, phorbol ester and LPS stimulated procoagulant activity was determined
Mori et al., Gut 1999 : The LPS induced increase in CAM expression was not significantly affected by prior treatment with either MTX or 6-MP