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FGFR3 — PGF

Pathways - manually collected, often from reviews:

• WikiPathways Focal Adhesion-PI3K-Akt-mTOR-signaling pathway: EFNA1/FGF1/FGF11/FGF10/EFNA2/EGF/FGF12/CSF1/ANGPT4/ANGPT2/ANGPT1/VEGFA/EFNA3/EFNA4/EFNA5/FGF14/FGF19/FGF17/FGF18/FGF2/FGF3/FGF4/FGF6/FGF7/FGF8/FGF9/FIGF/HGF/IGF1/INS/INS/KITLG/VEGFC/VEGFB/PDGFB/PGF/PDGFA/NGF/PDGFC/FGF21/FGF22/PDGFD/FGF20/FGF16 → FGFR2/KDR/INSR/FGFR3/IGF1R/KIT/FGFR1/EPHA2/EGFR/CSF1R/FGFR4/FLT1/FLT4/NGFR/MET/PDGFRA/PDGFRB/TEK (activation)
  

Text-mined interactions from Literome

Liu et al., Br J Pharmacol 1992 : ACh ( 30 microM ) induced a 1 +/- 1 %, 92 +/- 9 %, 62 +/- 5 % and 51 +/- 6 % reduction of the PGF2 alpha generated tension in neonatal, 3-10 days, 3-8 weeks and adult groups, respectively
Blanco-Rivero et al., Hypertension 2005 (Hypertension) : In SHR, ACh only increased the 6-keto-PGF1alpha production ( P < 0.05 )
Hsu et al., Phytomedicine 2006 (Dysmenorrhea) : The results show that DQSYS has the antagonistic action on both PGF ( 2 ) alpha and Ach induced uterine contraction
Chen et al., Placenta 1993 : We investigated the effect of an inhibitor of acetylcholine (ACh) synthesis, ( 2-benzoylethyl ) trimethylammonium iodide ( BETA ), on prostaglandin ( PG ) E2 and PGF2 alpha release from incubated placental explants in the presence or absence of ACh or arachidonic acid ( AA )
Kan et al., Mol Pharmacol 1996 : The PKC inhibitor calphostin C attenuated the ACh induced increase in PKC activity but not 6-keto-PGF1 alpha production ... These data suggest that ACh stimulates prostacyclin synthesis by activation of cPLA2 in a PKC independent mechanism and that both cPLA2 translocation to nuclear envelope and phosphorylation by MAPK are required for ACh induced 6-keto-PGF1 alpha synthesis in CEC
Kan et al., J Pharmacol Exp Ther 1997 : The receptor operated Ca++ channel blocker SK & F96365, but not the voltage dependent Ca++ channel blockers verapamil or nifedipine, attenuated ACh induced 6-keto-PGF1alpha production and the associated rise in cytosolic Ca++ ... In permeabilized CEC, guanosine 5'-O- ( 3-thiotriphosphate ) ( GTP-gamma-S ) but not ACh enhanced 6-keto-PGF1alpha synthesis ... ACh increased 6-keto-PGF1alpha production in the presence of GTP-gamma-S ... The NO synthase inhibitor N-monomethyl-arginine also failed to alter ACh induced 6-keto-PGF1alpha synthesis