Gene interactions and pathways from curated databases and text-mining

◀ Back to MAPK3

AGTR2 — MAPK3

Text-mined interactions from Literome

Zimpelmann et al., American journal of physiology. Renal physiology 2001 : In summary, AT(2) receptors in proximal tubule cells inhibit MAPK activity and stimulate phosphotyrosine phosphatase
Sekine et al., Arch Biochem Biophys 2003 : AT II also activated the extracellular signal regulated kinase ( ERK ) and p38 MAPK in the presence of AT II
Kumar et al., J Cardiovasc Pharmacol Ther 2003 (Disease Models, Animal...) : Our aim was to assess the effect of selective AT1 and AT2 receptor blockade with losartan and PD123,319, respectively, on MAPK expression after ischemia-reperfusion in isolated working rat hearts
Plouffe et al., Endocrinology 2006 : In summary, the present results demonstrate that AT(2) receptor induced sustained activation of p42/p44(mapk) and corresponding neurite outgrowth are mediated by phosphorylation of the nerve growth factor TrkA receptor
Guimond et al., Mol Cell Neurosci 2010 : Fyn is involved in angiotensin II type 2 receptor induced neurite outgrowth, but not in p42/p44mapk in NG108-15 cells ... Thus, although SKF activity is required to achieve AT(2) induced activation of TrkA, Rap1 and p42/p44(mapk) , Fyn is essential for AT(2) receptor induced neurite outgrowth, but not in AT(2) signaling leading to p42/p44(mapk) activation
Li et al., American journal of physiology. Renal physiology 2012 (MAP Kinase Signaling System) : We conclude that intracellular ANG II may induce NHE3 expression and activation in mPCTs via AT ( 1a ) - and AT(2) receptor mediated activation of MAP kinases ERK 1/2 and NF-?B signaling pathways
Hayashida et al., J Biol Chem 1996 : The antagonistic effect of the AT2-3LP on mitogen activated protein kinase activity and DNA synthesis were reversed by pertussis toxin and sodium orthovanadate
Shenoy et al., Endocrinology 1999 : Concurrent activation of c-Jun N-terminal kinase (JNK) and inhibition of Erk mitogen activated protein kinase activities is important for apoptosis in many cells, and we previously demonstrated that stimulation of AT2 receptors causes decreased mitogen activated protein kinase activity in neurons cultured from newborn rat hypothalamus and brain stem
McNeill et al., Endocr Res 1998 : Therefore, MAPK is located in the glomerulosa, where the AT1 receptor is localised and concerned with aldosterone biosynthesis, and in the medulla where MAPK activation results from AT2R activation