Gene interactions and pathways from curated databases and text-mining

◀ Back to CA2

CA2 — TET3

Text-mined interactions from Literome

Wu et al., Life Sci 2001 : We found that Tet inhibited both electrically induced [Ca2+ ] i transient and protein release during the Ca2+ paradox, while Fan had no significant effects
Wang et al., Acta Pharmacol Sin 2002 : To study the effects of tetrandrine ( Tet ) on extracellular Ca2+ influx and intracellular Ca2+ release in cultured calf basilar artery smooth muscle cells
Leung et al., Br J Pharmacol 1994 (Leukemia, Promyelocytic, Acute) : 2. Pretreatments with 100 nM rauwolscine ( alpha 2-adrenoceptor antagonist ), 100 nM prazosin ( alpha 1-adrenoceptor antagonist ), 10 nM phorbol myristate acetate ( PMA, a protein kinase C activator ) or 100 nM staurosporine ( a protein kinase C inhibitor ) had no effect on 100 microM TET induced intracellular Ca2+ release ... 5. These data suggest that ( a ) TET and TSG mobilized the same Ca2+ pool and TET induced intracellular Ca2+ release was independent of protein kinase C activity and ox-adrenoceptor activation, and ( b ) TET blocked the voltage-insensitive Ca2+ entry pathway activated by TSG
Zeng et al., Zhongguo Yao Li Xue Bao 1993 : Tet 30 mumol.L-1 depressed norepinephrine 10 mumol.L-1 induced [Ca2+ ] i elevation by 42 %
Deng et al., Yao Xue Xue Bao 1993 : In addition, Tet was also found to inhibit the decrease of Ca2+ and the increase of Na+ in myocardial tissues during calcium-free perfusion
Yao et al., Yao Xue Xue Bao 1995 (Myocardial Reperfusion Injury) : Tet also attenuated Ca2+ accumulation in myocardial cell, reduced melondialdehyde ( MDA ) production in ischemic myocardial and decreased CPK release in comparison with the control
Leung et al., Clin Exp Pharmacol Physiol 1996 : The effects of TET and its closely related analogues, hernandezine ( HER ) and berbamine ( BER ), on Ca2+ entry and Ca2+ release were compared in fura-2 loaded HL-60 cells
Takemura et al., Clin Exp Pharmacol Physiol 1996 : 5. Our results suggest that the action of TET on Ca2+ entry is dependent on cell types and that TET inhibits both Ca2+ entry from the extracellular medium and Ca2+ release from intracellular stores in rat glioma C6 cells
Viviani et al., Toxicol Appl Pharmacol 1996 : We have investigated the effects of triethyltin ( TET ) on agonist stimulated Ca2+ signaling and neurosecretion in PC12 cells ... TET potentiated the peak Ca2+ response stimulated by Bk in both the presence and the absence of extracellular Ca2+, and prolonged the recovery phase after ATP stimulation
Imoto et al., Res Commun Mol Pathol Pharmacol 1997 (Glioma) : TET and HER alone did not affect the resting cytoplasmic Ca2+ concentration ([Ca2+]i) ... The present results suggest that TET and HER inhibit Ca2+ release from intracellular stores as well as Ca2+ entry from extracellular medium evoked by bombesin and TG