◀ Back to ICAM1
CD44 — ICAM1
Protein-Protein interactions - manually collected from original source literature:
Studies that report less than 10 interactions are marked with *
-
STRING interaction:
ICAM1
—
CD44
(interaction, mapped from kegg_pathways)
-
STRING interaction:
CD44
—
ICAM1
(interaction, mapped from kegg_pathways)
Text-mined interactions from Literome
Chute et al., Exp Hematol 1999
:
Despite the high level of cellular activation and proliferation induced by PMVEC coculture, the surface expression of adhesion molecules CD11a ( LFA-1 ), CD11b, CD15s ( sialyl-Lewis x ), CD43, and
CD44 ( HCAM ) on the total CD34+ population was maintained, and the surface expression of CD49d ( VLA-4 ),
CD54 ( ICAM ), CD58, and CD62L ( L selectin )
increased after ex vivo expansion ... Despite the high level of cellular activation and proliferation induced by PMVEC coculture, the surface expression of adhesion molecules CD11a ( LFA-1 ), CD11b, CD15s ( sialyl-Lewis x ), CD43, and
CD44 ( HCAM ) on the total CD34+ population was maintained, and the surface expression of CD49d ( VLA-4 ), CD54 (
ICAM ), CD58, and CD62L ( L selectin )
increased after ex vivo expansion
De Saint Jean et al., Exp Eye Res 2004
:
In the primary cultured cells, TNFalpha
induced an important upregulation of
ICAM-1 , Fas and CD40 whereas
CD44 and CD63 were significantly decreased
Patel et al., J Clin Immunol 1995
:
While activation of TE cells with IFN-gamma for 48 hr induced a greater than fivefold increase in the expression of four surface molecules ( CD38,
CD54 , MHC class I, and MHC class II ), it also
induced a greater than 50 % increase in the expression of 14 other surface molecules ( CD12, CD29, CD40,
CD44 , CD47, CD49b, CD49c, CD49e, CD55, CD66, CD87, CD104, TE4, and STE3 ) and a decrease in the expression of three molecules ( CDw65, CDw109, and STE2 )
Hawley et al., Clin Exp Metastasis 1993
(Neoplasm Metastasis...) :
Despite higher ICAM-1 levels, cell aggregation assays revealed that
LFA-1-ICAM-1 adhesive interactions were not
involved in the homotypic adhesion of B9/BM1 cells but rather that binding of
CD44 to endogenously synthesized hyaluronan was responsible