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FGF3 — FGF8

Pathways - manually collected, often from reviews:

• Reactome Reaction: FGF3 → FGF8 (reaction) Carpenter et al., Exp Cell Res 1999, Wong et al., Proc Natl Acad Sci U S A 2002, Lax et al., Mol Cell 2002, Fong et al., J Biol Chem 2003, Agazie et al., Oncogene 2003, Takeda et al., Clin Cancer Res 2007, Ahmed et al., Biochem J 2008, Schüller et al., Biochem J 2008, Qing et al., J Clin Invest 2009, Turner et al., Nat Rev Cancer 2010, Bai et al., Cancer Res 2010, Dutt et al., PloS one 2011, Wesche et al., Biochem J 2011, Klint et al., J Biol Chem 1995, Wang et al., Mol Cell Biol 1994, Ong et al., Biochem Biophys Res Commun 1996, Kanai et al., J Biol Chem 1997, Kouhara et al., Cell 1997, Ong et al., Biochem Biophys Res Commun 1997, Hadari et al., Mol Cell Biol 1998, Raffioni et al., J Biol Chem 1998

Text-mined interactions from Literome

Wilson et al., Curr Biol 2000 : Fgf3 was also found to be expressed in the early epiblast, and ongoing FGF signalling in epiblast cells was required for acquisition of neural fate and for the suppression of Bmp4 and Bmp7 expression
Kettunen et al., Dev Dyn 2000 : In vitro analyses showed that expression of Fgf-3 and Fgf-10 in the dental mesenchyme was dependent on dental epithelium and that epithelially expressed FGFs, FGF-4 and -8 induced Fgf-3 but not Fgf-10 expression in the isolated dental mesenchyme
Liu et al., Development 2002 (Limb Deformities, Congenital) : In this system, FGF10 but not FGF8 protein injected into the mutant distal tip mesenchyme restores Fgf8 expression in the AER
Saitsu et al., Mech Dev 2006 (Heart Defects, Congenital) : Moreover, over-expression of Fgf15 resulted in up-regulation of Fgf8 expression in the isthmus/r1
Katayama et al., Int J Oncol 2010 (Prostatic Neoplasms) : Overall, bicalutamide inhibits the cyclin A expression possibly by inhibiting FGF-8 mRNA expression and FGF-8 protein secretion but not by inhibiting FGF receptor ( FGFR ) signalling in androgen dependent cell lines, and by other mechanisms in androgen independent cell lines
Pearson et al., Development 2011 : We show that collar cells are composed of Fgf3 ( + ) SOX3 ( + ) proliferating progenitors, the induction of which is SHH dependent, but the maintenance of which requires FGF signalling
Vendrell et al., Mech Dev 2013 (Wnt Signaling Pathway) : Interestingly however, Wnt8a and Fgf3 are redundantly required for expression of Fgf15 in the hindbrain indicating additional reciprocal interactions between Fgf and Wnt signalling
Miyake et al., Biology open 2013 : fgf3 and fgf8 were expressed earlier than fgf22 in the MHB primordium and Fgf3/Fgf8 signaling was required for fgf22 expression in the posterior midbrain