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CA2 — CMC2
Text-mined interactions from Literome
Kabbara et al., Cell Calcium 1999
:
The magnitude of the peak of the
4-CmC induced
Ca2+ transient was not significantly changed by removal of extracellular Ca2+ nor by inhibiting the SR Ca2+ pump with 2,5-di-tert-butylhydroquinone
Tupling et al., J Appl Physiol 2002
(Ion Channel Gating) :
Both AgNO3 and
4-CMC fully
inhibited Ca2+-ATPase activity measured in homogenates ... These findings indicate that AgNO3, but not
4-CMC ,
induces Ca2+ release by acting on both the CRC and the Ca2+-ATPase
Fessenden et al., J Biol Chem 2003
:
To identify region ( s ) on RyR1 that are responsible for mediating activation by 4-CmC, we expressed RyR1-RyR3 chimeric proteins in dyspedic 1B5 myotubes and then measured
4-CmC induced increases in intracellular
Ca2+
Zhao et al., Biophys Chem 2008
:
CAC is independent of DNA concentration, but the
CMC of the mixed solutions of DNA and 12-3-12 ( CMC ( mix ) )
increases with the increasing of DNA concentration
Herrmann-Frank et al., Biochim Biophys Acta 1996
:
In heavy SR vesicles obtained from rabbit back muscles,
4-CmC stimulated (
Ca2+ ) -activated [ 3H ] ryanodine binding with an EC50 of about 100 microM ... Since these data strongly indicate that
4-CmC specifically
activates SR
Ca2+ release also in intact cell systems, this substance might become a powerful tool to investigate ryanodine receptor mediated Ca2+ release in muscle and non-muscle tissue
Westerblad et al., Cell Calcium 1998
:
4-CmC potentiated the
[Ca2+ ] i increase and force of contraction induced by tetanic stimulation
Struk et al., J Physiol 1999
(Calcium Signaling) :
6. The stimulating
effect of 5 microM
4-CmC on depolarization induced
Ca2+ release resembled the potentiation by 0.5 mM caffeine ... 7. Neither 5 microM
4-CmC nor 0.5 mM caffeine
led to a loss of voltage control of
Ca2+ release during repolarization after short depolarizations, as has been reported previously for caffeine