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CDKN1A — STAT5A

Text-mined interactions from Literome

Kawashima et al., J Immunol 2001 (Leukemia, Myeloid, Acute) : We recently demonstrated that STAT5 can induce a variety of biological functions in mouse IL-3 dependent Ba/F3 cells ; STAT5 induced expression of pim-1, p21 ( WAF/Cip1 ), and suppressor of cytokine signaling-1/STAT induced STAT inhibitor-1/Janus kinase binding protein is responsible for induction of proliferation, differentiation, and apoptosis, respectively
Brizzi et al., Diabetes 2002 (Diabetes Mellitus) : Collectively, our results provide evidence for the role of glycation in dm-LDL mediated effects and for a specific role of src kinase in STAT5 dependent p21 ( waf ) expression
Brizzi et al., FASEB J 2004 (Albuminuria...) : The role of STAT5 in AGE- and TGF-beta mediated p21waf expression and growth arrest, but not collagen production, was confirmed by the expression of the dominant negative STAT5 ( DeltaSTAT5 ) or the constitutively activated STAT5 ( 1*6-STAT5 ) constructs
Mallette et al., J Biol Chem 2007 : However, ca-STAT5A did not induce p21 and p16(INK4a), which are responsible for inhibiting cyclin dependent protein kinases and engaging the Rb pathway during the senescence response to oncogenic ras
Matsumura et al., Mol Cell Biol 1997 (Leukemia, Megakaryoblastic, Acute) : Thrombopoietin induced differentiation of a human megakaryoblastic leukemia cell line, CMK, involves transcriptional activation of p21 ( WAF1/Cip1 ) by STAT5