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JUN — TAT

Text-mined interactions from Literome

Lim et al., J Virol 2000 (Astrocytoma) : We postulate that a Tat mediated increase in SP1 binding activities augments the binding of AP1 and NF-kappaB, leading to synergistic activation of the MCP-1 promoter
Gibellini et al., Br J Haematol 2001 : The Tat mediated induction of c-fos and AP-1 in uninfected lymphoid T cells may contribute to explain the immune hyperactivation that characterizes the progression to autoimmuno deficiency syndrome and constitutes the optimal environment for HIV-1 replication, occurring predominantly in activated/proliferating CD4+ T cells
Lee et al., Cardiovasc Res 2004 : However, Tat did not affect DNA binding activities of AP-1 , CREB, and STAT1
Guan et al., Neuroscience 2006 (Brain Injuries...) : As a result, Tat-JBD diminished the increased phosphorylation of c-Jun ( a nuclear substrate of JNK ) and the increased expression of Fas ligand induced by ischemia/reperfusion in the vulnerable hippocampal CA1 subregion
Hidalgo-Estévez et al., J Gen Virol 2006 : Constitutive or transient Tat expression in Jurkat T cells enhanced cooperative NFAT/AP-1- but not AP-1 dependent transcription independent of its ability to transactivate the HIV-1 LTR