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ICAM3 — IRF6
Text-mined interactions from Literome
Chakravortty et al., Biochim Biophys Acta 1999
:
LPS induced
ICAM expression in lamina propria fibroblasts was modulated by whole blood, PBMC and neutrophils
Banu et al., Exp Nephrol 2002
:
These studies demonstrate normal patterns of class II MHC,
ICAM-1 , and B7 expression in TGF-beta1 ( -/- ) tubular epithelial cells in
response to IFN-gamma,
LPS , and TGF-beta1
Myers et al., Am J Physiol 1992
:
We conclude from these data that although PMA induced ICAM-1 expression may be triggered through activation of protein kinase C,
ICAM-1 induction by IL-1 beta, TNF-alpha, or
LPS may involve distinct regulatory pathway ( s )
Mazzola et al., FASEB J 2005
(Disease Models, Animal...) :
Lastly, CO pretreatment prevents
LPS induced
ICAM expression on lung endothelium and inhibits leukocyte marginalization on lung parenchyma
Waldow et al., Nitric Oxide 2006
:
The
induction of vascular cell adhesion molecule ( VCAM ),
intercellular adhesion molecule ( ICAM ) and E-selectin by the cytokines TNFalpha and IL-1beta, and by bacterial
lipopolysaccharide (LPS) was reduced by SNAP/Cys preincubation ( 30 min, 1mM ) to less than 10 % of controls
Singh et al., Phytomedicine 2008
:
The major CAMs, viz.,
intercellular adhesion molecule-1 ( ICAM-1 ), VCAM-1 and E-selectin are expressed on endothelium in
response to various cytokines or bacterial
LPS
Liu et al., Chin Med J (Engl) 2010
(Reperfusion Injury) :
After treatment with 2 L/min of 250 ppm CO i.p., the increase of PAF,
ICAM-1 , MDA, MPO, and cell apoptotic rate
induced by
LPS was markedly reduced ( P < 0.05 or 0.01 ), and accompanied by ameliorating intestine injury
Han et al., BMC complementary and alternative medicine 2011
(Disease Models, Animal...) :
Treatment with FSE significantly inhibited
LPS induced increases in IL-1ß and TNF-a production and the expression of iNOS,
ICAM-1 and COX-2
Qureshi et al., Lipids in health and disease 2011
(Inflammation...) :
The microarray DNA analyses, followed by pathway analyses indicated that quercetin or d-tocotrienol inhibit several
LPS induced expression of several ageing and pro-inflammatory genes ( IL-1ß, IL-1a, IL-6, TNF-a, IL-12, iNOS, VCAM1,
ICAM1 , COX2, IL-1RA, TRAF1 and CD40 )
Han et al., Inflammation 2012
(Inflammation...) :
AGF at the dose level of 500 mg/kg significantly inhibited
LPS induced upregulation of NF-?B activation and the production of IL-1ß, TNF-a, iNOS,
ICAM-1 , and COX-2
Joo et al., Gut and liver 2012
:
U0126 ( an inhibitor of MEK-1/2 ) suppressed the
LPS induced IL-12p40, IL-6, MCP-1,
ICAM-1 , and VCAM-1 mRNA accumulation in BMMs
Repo et al., Scand J Immunol 1994
:
Alkali treated
LPS of Yersinia enterocolitica does not
induce expression of E-selectin,
ICAM-1 or VCAM-1 on endothelial cells but may mediate antibody- and complement dependent cell injury
Goebeler et al., Immunobiology 1993
(Inflammation) :
Furthermore,
ICAM-1 expression by bone marrow derived M phi could be
up-regulated by tumor necrosis factor-alpha, interferon-gamma and
LPS
Sakai et al., Life Sci 1996
:
Pretreatment of diclofenac inhibited
LPS induced expression of E-selectin,
intercellular adhesion molecule-1 ( ICAM-1 ) and vascular cell adhesion molecule-1 ( VCAM-1 ) in HUVEC, determined by flow cytometry and a cellular enzyme linked immunosorbent assay ( cell-ELISA ) ... The inhibitory activity was concentration dependent between 15.6 and 250 micrograms/ml. Diclofenac also inhibited
LPS induced increases in E-selectin,
ICAM-1 and VCAM-1 mRNAs, indicating that the action of diclofenac is to inhibit synthesis of these molecules
Fujisawa et al., J Dermatol Sci 1997
(Carcinoma, Squamous Cell) :
This evidence contrasts with previously reported findings indicating that, in human cultured keratinocytes,
ICAM-1 expression is
induced by IFN-gamma and TNF-alpha, and not by either IL-1 or
LPS
Lo et al., J Immunol 1998
:
FACS analysis reveals that LPG treatment blocked the
LPS mediated expression of E-selectin,
intercellular adhesion molecule-1 , and vascular cell adhesion molecule-1 on endothelial cells and monocyte adhesion without altering the integrity of the endothelial monolayer