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ICAM1 — IL10

Text-mined interactions from Literome

Buchwald et al., Eur J Med Res 1999 : We studied the effects of IL-10 on important functions of native and granulocyte-macrophage colony stimulating factor ( GM-CSF ) activated neutrophils and monocytes, namely phagocytosis and membrane expression of the beta superset2-integrins and of the intercellular adhesion molecule-1 ( ICAM-1 )
Kooy et al., J Pathol 1999 (Carcinoma, Basal Cell...) : Interferon-gamma induced ICAM-1 and CD40 expression, complete lack of HLA-DR and CD80 ( B7.1 ), and inconsistent HLA-ABC expression in basal cell carcinoma : a possible role for interleukin-10 ? ... It may be concluded that it is highly likely that the presence of IL-10 in BCC is directly or indirectly responsible for the complete lack of expression of HLA-DR, ICAM-1 , CD40 and CD80 and the inconsistent expression of HLA-ABC on BCC cells in situ and may be a way of escaping immune survillance
Fukatsu et al., Arch Surg 1999 : The levels of intestinal interleukin 10 and interleukin 4, inhibitors of intercellular adhesion molecule-1 ( ICAM-1 ) expression, decline with total parenteral nutrition (TPN)
Wirjatijasa et al., J Neurosci Res 2002 : In summary, we demonstrate that IL-4, IL-10 , and IL-1-ra induce microglial ramification and reduce ICAM-1-expression , whereas the secretion of proinflammatory cytokines is not prevented
Salek-Ardakani et al., Virology 2002 : Viral IL-10 in a concentration dependent manner inhibited both basal- and IFN-gamma induced HLA-class II, ICAM-1 ( basal levels of ICAM-2 and ICAM-3 is unaffected ), CD80, and CD86 up-regulation when added simultaneously with IFN-gamma ... Viral IL-10 in a concentration dependent manner inhibited both basal- and IFN-gamma induced HLA-class II, ICAM-1 ( basal levels of ICAM-2 and ICAM-3 is unaffected ), CD80, and CD86 up-regulation when added simultaneously with IFN-gamma
Yang et al., Hua Xi Kou Qiang Yi Xue Za Zhi 2002 (Gingivitis) : [ Effects of exogenous IL-10 on IL-6 and ICAM-1 expression in inflammatory gingival tissue ]
Giavazzi et al., Cancer Res 1992 (Melanoma) : Tumor necrosis factor, interleukin 1, and gamma-interferon also caused the release of soluble ICAM-1
Andersen et al., Nephrol Dial Transplant 1992 : Furthermore, the in vitro model indicates that ICAM-1 expression is regulated by gamma-interferon and interleukin-1 produced by activated T lymphocytes and macrophages
Stuyt et al., Immunology 2003 : Selective regulation of intercellular adhesion molecule-1 expression by interleukin-18 and interleukin-12 on human monocytes
Furbert-Harris et al., Cell Mol Biol Incl Cyto Enzymol 2003 (Neoplasm Metastasis...) : ICAM-1 , expressed on PC-3 and DU 145 cells, was enhanced by TNF-alpha and IL-10
Cawood et al., Eur J Endocrinol 2006 (Graves Ophthalmopathy) : Tumour necrosis factor-alpha (TNFalpha) and interleukin1 (IL1) ( 0.1 ng/ml ) stimulated ICAM1 expression by eight- to ten-fold
Moreira et al., Am J Pathol 2006 (Disease Susceptibility...) : Expression of ICAM-1 was up-regulated on T lymphocytes after infection with the fungus, and its expression was dependent on interferon-gamma, tumor necrosis factor-alpha, and interleukin-12
Akiyoshi et al., Transplantation 2006 : SH induced indefinite survival of fully allogeneic cardiac allografts, generated CD4 regulatory cells, inhibited ICAM-1 expression in the allografts, and upregulated IL-4 and IL-10 production
Krutmann et al., J Invest Dermatol 1990 : Stimulation of cells with recombinant human ( rh ) interleukin (IL) 1 alpha, rhIL-4, rhIL-5, rhIL-6, rh granulocyte/macrophage colony stimulating factor ( GM-CSF ), rh interferon alpha ( rhIFN alpha ), and rh transforming growth factor beta ( TGF beta ) did not increase ICAM-1 surface expression
Dace et al., PloS one 2009 (Demyelinating Diseases...) : IL-10 upregulates ICAM-1 within neural tissues, promoting massive macrophage influx, inflammation induced demyelination, and subsequent loss of neural tissue resulting in muscle weakness and paralysis
Giwa et al., Neurology 2012 (Cerebral Small Vessel Diseases) : Endothelial dysfunction is a possible causal factor, and circulating markers of endothelial activation ( intercellular adhesion molecule-1 , thrombomodulin ) and inflammation ( interleukin [ IL]-6 ) are elevated in patients with SVD
Schulz et al., Blood 2013 (Leukemia, Lymphocytic, Chronic, B-Cell) : The increase in IL-10 levels lead to an induction of STAT1 phosphorylation in CLL cells and to enhanced cell-surface expression of intercellular adhesion molecule 1 and altered expression of cytoskeletal and migration related genes
Willems et al., Eur J Immunol 1994 : Interleukin-10 inhibits B7 and intercellular adhesion molecule-1 expression on human monocytes ... The inhibitory effect of IL-10 both on ICAM-1 and B7 expression was shown to be dose dependent
Sano et al., Mol Cell Biochem 1994 : Cellular expression of ICAM-1 was drastically induced by TNF or interleukin-1 stimulation, and the moderate expression with delayed-action was observed only by lipopolysaccharide stimulation
Ranheim et al., Cell Immunol 1995 : Tumor necrosis factor-alpha facilitates induction of CD80 ( B7-1 ) and CD54 on human B cells by activated T cells : complex regulation by IL-4, IL-10 , and CD40L
Krakauer et al., Immunol Lett 1995 : IL-10 also down-regulated the expression of ICAM-1 and VCAM-1 on IL-1 activated endothelial cells
Shrikant et al., J Immunol 1995 : These results suggest that IL-10 affects ICAM-1 expression at the translational and/or post-translational level ... Thus, both IL-10 and IL-6 can inhibit ICAM-1 expression by glial cells, although they do so by contrasting mechanisms
Spittler et al., Immunology 1995 : The expression of intercellular adhesion molecule-1 ( ICAM-1 ) /CD54 on monocytes was significantly inhibited by IL-10
Wake et al., Blood 1995 (Bone Resorption...) : We report the following novel features of homotypic adhesion via leukocyte function associated antigen-1 ( LFA-1 ) /intracellular adhesion molecule-1 ( ICAM-1 ) pathway that suggest a role for it in cytokine production and rapid proliferation of ATL cells : ( 1 ) ATL cells show clustering in a calcium dependent manner, even at the higher concentration ; ( 2 ) ATL cells consistently and highly express ICAM-1 and an active form of LFA-1, whereas integrin expression, except for LFA-1, is rather lower compared with that of normal CD4+ T cells ; ( 3 ) ATL cells make conjugate formation within 6 minutes and clustering within 48 hours, both of which are inhibited by the addition of monoclonal antibodies ( MoAbs ) against LFA-1 and ICAM-1 ; ( 4 ) spontaneous mRNA transcription and protein secretion of both interleukin-1 and parathyroid hormone related protein are observed consistently in ATL cells, and these productions are inhibited by anti-LFA-1 and anti-ICAM-1 MoAbs but are markedly increased by cross linking of LFA-1 and ICAM-1 by the immobilized specific MoAbs ; and ( 5 ) proliferative responses of ATL cells are also inhibited by these MoAbs
Campbell et al., J Urol 1994 (Carcinoma, Transitional Cell...) : In contrast, interleukin-1 and phorbol myristate acetate exhibited variable effects on ICAM-1 expression, and interferon-alpha had no effect
Shrikant et al., J Neuroimmunol 1994 (Encephalomyelitis, Autoimmune, Experimental) : Regulation of intercellular adhesion molecule-1 gene expression by tumor necrosis factor-alpha, interleukin-1 beta, and interferon-gamma in astrocytes
Boorstein et al., Ophthalmology 1994 (Keratitis, Herpetic) : In this study, the authors investigated the effects of interleukin-10 (IL-10) on HLA-DR and ICAM-1 expression in human HSK
Chang et al., Eur J Immunol 1994 : IL-10 potently inhibits the TNF-alpha induced ICAM-1 up-regulation, but not the IFN-gamma induced H-2K up-regulation
Deisher et al., Life Sci 1993 (Second Messenger Systems) : Surface protein expression of vascular cell adhesion molecule-1, endothelial leukocyte adhesion molecule-1, or intercellular adhesion molecule-1 , which is induced by tumor necrosis factor, interleukin-1 , and lipopolysaccharide, was not induced by pentoxyfilline, a phosphodiesterase inhibitor, nor by dibutyryl cyclic adenosine monophosphate
Lai et al., Int J Cancer 1993 (Fibrocystic Breast Disease) : Possible regulation of soluble ICAM-1 levels by interleukin-1 in a sub-set of breast cysts
Baumgartner-Parzer et al., Diabetologia 1995 : Stimulation of confluent HUVECs, kept in 30 vs 5 mmol/l glucose for 13 +/- 1 days, with 20 U/ml interleukin-1 for 24 h ( ICAM-1 ) and 4 h ( endothelial leukocyte adhesion molecule 1 ) resulted in reduced ICAM-1 ( 84.8 +/- 27.0 %, p < 0.05 ) and endothelial leukocyte adhesion molecule-1 ( 87.6 +/- 22.4 %, p < 0.05 ) expression vs control cells, while that of PECAM ( t : 24 h ) and vascular cell adhesion molecule-1 ( t : 16 h ) remained unchanged
Perretti et al., Biochem Biophys Res Commun 1996 : This opposite modulatory role of interleukin-1 and dexamethasone on intercellular adhesion molecule-1 expression was also, for the first time, observed in vivo using mouse peritoneal macrophages : a four-fold increase in intercellular adhesion molecule-1 expression was measured after local administration of the cytokine ( 5 micrograms/kg ) and this effect was greatly inhibited ( > 70 % ) by co-injection with 1 microgram dexamethasone
Ikeda et al., J Am Soc Nephrol 1996 : In an enzyme linked immunosorbent assay, interleukin 1 beta ( IL-1 beta ; 10 ng/mL ) increased ICAM-1 molecule expression on cultured rat mesangial cell surface in a time dependent manner
Song et al., Blood 1997 : Interleukin-10 inhibits interferon-gamma induced intercellular adhesion molecule-1 gene transcription in human monocytes ... IL-10 inhibition of IFN-gamma induced ICAM-1 expression was apparent as early as 3 hours and was blocked by an anti-IL-10 antibody but not by an isotype matched control antibody ... Thus, IL-10 inhibits IFN-gamma induced ICAM-1 expression in monocytes primarily at the level of gene transcription ... These data indicate that IL-10 inhibits IFN-gamma induced transcription of the ICAM-1 gene by a regulatory mechanism that may involve NF-kappaB
Paolieri et al., Allergy 1997 : IL-4, IL-6, IL-8, IL-10 , and TGF-beta 1 did not affect either ICAM-1 expression or sICAM-1 release
Wang et al., Circ Res 1998 : Cardiac graft intercellular adhesion molecule-1 ( ICAM-1 ) and interleukin-1 expression mediate primary isograft failure and induction of ICAM-1 in organs remote from the site of transplantation
Labuda et al., Immunology 1998 : ICAM-1 costimulation induces IL-2 but inhibits IL-10 production in superantigen activated human CD4+ T cells ... A major finding in the present study was that ICAM-1 costimulation strongly inhibits IL-10 production in CD4+ T cells
Chatelain et al., Arch Dermatol Res 1998 : IL-10 inhibits ICAM-1 expression on human Langerhans cells but not on keratinocytes, dermal endothelial cells or fibroblasts