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CNR1 — NFKB1

Text-mined interactions from Literome

Sancho et al., Mol Pharmacol 2003 : The NF-kappaB inhibitory activity of AEA was independent of CB(1) and CB(2) activation in TNFalpha stimulated 5.1 and A549 cell lines, which do not express vanilloid receptor 1, and was not mediated by hydrolytic products formed through the activity of the enzyme fatty acid amide hydrolase
Jüttler et al., Neuropharmacology 2004 : This suggests that neither the antioxidant properties of cannabinoids nor binding to CB1 or CB2 receptors are responsible for the inhibition of NF-kappaB activity and apoptosis