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AHR — PDCD5
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Hardin et al., Toxicol Appl Pharmacol 1992
:
The results support the hypothesis that B [ a ] P suppression of B cell lymphopoiesis is mediated at least in part by the induction of
programmed cell death and that the
Ah receptor and/or P450 isoenzymes are
involved in this process
Vogel et al., Am J Pathol 2007
(Lymphoma) :
Here, we show that activation of the
aryl hydrocarbon receptor (AhR) by 2,3,7,8-tetrachlorodibenzo-p-dioxin ( TCDD )
resulted in loss of the
programmed cell death ( apoptosis ) response in three different lymphoma cell lines, which plays a key role in the development of cancer, especially lymphoma and leukemia
Chopra et al., Crit Rev Toxicol 2011
:
Dioxin toxicity,
aryl hydrocarbon receptor signaling, and apoptosis-persistent pollutants
affect programmed cell death