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FOS — IL7
Text-mined interactions from Literome
Seppänen et al., Oncol Res 1998
(Adenocarcinoma...) :
In the present study, we have investigated the
effects of interferons-alpha (IFN-alpha) and -gamma ( IFN-gamma ),
interleukin-10 (IL-10) and -13 ( IL-13 ), transforming growth factor-beta1 ( TGF-beta1 ), granulocyte-macrophage colony stimulating factor ( GM-CSF ), and tumor necrosis factor-alpha (TNF-alpha) on cell proliferation and induction of transcription factors
AP-1 and NF-kappaB in UM-EC-3 human endometrial adenocarcinoma cells and UT-OC-5 ovarian carcinoma cells in vitro
Liacini et al., Matrix Biol 2002
(Osteoarthritis, Hip) :
Inhibition of
interleukin-1 stimulated MAP kinases,
activating protein-1 (AP-1) and nuclear factor kappa B (NF-kappa B) transcription factors down-regulates matrix metalloproteinase gene expression in articular chondrocytes
Wang et al., Nat Immunol 2006
:
Tumor necrosis factor receptor associated factor 6 ( TRAF6 ) is critical for mediating Toll-like receptor ( TLR )
-interleukin 1 receptor (IL-1R) signaling and subsequent
activation of NF-kappaB and
AP-1 , transcriptional activators of innate immunity
Ming et al., Cancer Immunol Immunother 2012
(Carcinoma, Non-Small-Cell Lung...) :
We found that, in lung cancer cell lines and in nude mice,
Interleukin-7/Interleukin-7 receptor increased the expression of cyclin D1 and phosphorylation of c-Fos/c-Jun,
induce c-Fos and c-Jun heterodimer formation, and enhanced c-Fos/c-Jun DNA binding activity to regulate cyclin D1
Ming et al., Zhonghua Bing Li Xue Za Zhi 2012
(Carcinoma, Non-Small-Cell Lung...) :
IL-7/IL-7R could promote proliferation of A549 cell, increase cyclin D1 and VEGF-D expression, and
enhance c-Fos/c-Jun expression and phosphorylation, resulting in formation of heterodimer ... Furthermore,
IL-7/IL-7R could
induce binding of
c-Fos/c-Jun to cyclin D1/VEGF-D promoters and regulate their transcription
Sung et al., J Biol Chem 1993
:
Stimulation of
interleukin-1 gene transcription may be
caused by the stimulation of transcription factor activities, including those of
AP-1 , by these protein phosphatase inhibitors
Swiergiel et al., Brain Res Bull 1996
:
The role of cerebral noradrenergic systems in the
Fos response to
interleukin-1
Chang et al., Brain Res 1996
:
FOS expression
induced by
interleukin-1 or acute morphine treatment in the rat hypothalamus is attenuated by chronic exposure to morphine
Gringhuis et al., Blood 1997
(Leukemia-Lymphoma, Adult T-Cell) :
We also show that the
IL-7 induced enhancement of the
AP-1-DNA binding activity is not cyclosporin A-sensitive