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BAX — PCNA
Protein-Protein interactions - manually collected from original source literature:
Studies that report less than 10 interactions are marked with *
Text-mined interactions from Literome
Bossenmeyer-Pourié et al., Neuroscience 2002
(Hypoxia, Brain) :
Transient overexpression of
proliferating cell nuclear antigen was
followed by increasing expression of p53, p21,
Bax and caspases, whereas Bcl-2 and heat shock proteins were progressively repressed
Azrak et al., Clin Cancer Res 2004
(Carcinoma, Squamous Cell...) :
Sensitivity to this combination was associated with poor differentiation status, higher
cyclin A index, recruitment of cells into S phase, and
induction of
Bax expression and apoptosis
Castro et al., Am J Physiol Gastrointest Liver Physiol 2007
:
Cyclin D1 overexpression
increased DCA induced
Bax translocation, cytochrome c release, and apoptosis
Liu et al., Int J Cancer 2008
(Body Weight...) :
Proliferating cell nuclear antigen ( PCNA ), cyclin D1 and Bcl-2 expression decreased, and
Bax expression and nuclear fragmentation
increased with increasing dietary beta-ionone
Wang et al., J Asian Nat Prod Res 2009
:
1 in high concentrations
induced Bax , caspase-3, and cyclin dependent kinase (CDK) inhibitor P21 expression, whereas the expressions of cyclin CDK2, Bcl-2, and
proliferating cell nuclear antigen ( PCNA ) were decreased by 1 treatment
Nair et al., Hum Reprod 2013
(Leiomyoma...) :
Western blot analysis showed that, in comparison with the untargeted vector Ad-TK, Ad-SSTR-RGD-TK/GCV more effectively reduced expression of proteins that regulate the cell cycle ( Cyclin D1 ) and proliferation (
PCNA , Proliferating Cell Nuclear Antigen ), and it
induced expression of the apoptotic protein
BAX , in HuLM cells
Xu et al., PloS one 2013
:
Reduced hepatic damage was confirmed with histopathology in the sTNFR : IgG-Fc treated group, which is consistent with reduced Bcl-2 and
Bax , at mRNA and protein levels, but
increased hepatocyte proliferation (
PCNA )