Gene interactions and pathways from curated databases and text-mining

◀ Back to BDNF

BDNF — RBBP7

Text-mined interactions from Literome

Morita et al., J Neurosci Res 2009 : In addition, HDAC inhibitors enhanced serotonin stimulated BDNF gene expression, the enhancement of which could be abolished by the inhibition of 5alpha reduced neurosteroid production in the glioma cells
Almeida et al., Neurotox Res 2010 : Both NGF and BDNF counteracted the increase in the levels of histone H3 and H4 acetylations and reduced histone deacetylase (HDAC) activity induced by 3-NP
Wei et al., J Neuroimmunol 2010 : The results suggest that inhibition of HDAC activity and BDNF-TrkB autocrine loop are involved in regulation of astrocytic BDNF transcription, whereas the mechanisms for elevated constitutive gene BDNF expression of LEW/N astrocytes remain to be investigated
Tian et al., Ann N Y Acad Sci 2010 : Taken together, these findings suggest that inhibition of HDAC activity by TSA activates Bdnf transcription and a compensatory change in HDAC1 expression in neurons
Calfa et al., Hippocampus 2012 : HDAC activity is required for BDNF to increase quantal neurotransmitter release and dendritic spine density in CA1 pyramidal neurons
Zhang et al., Neuroscience 2012 (Disease Models, Animal) : These results demonstrate that VPA appears to protect RGCs from ONC by inhibiting neuronal apoptosis possibly via the activation of BDNF-TrkB signaling and HDAC inhibition
Soriano et al., J Neurochem 2013 : We found that, while HDAC5 and HDAC4 mutants lacking their N-terminal phosphorylation sites ( HDAC4 ( MUT ), HDAC5 ( MUT ) ) are constitutively nuclear, co-expression with SMRT renders them exportable by signals that trigger SMRT export, such as synaptic activity, HDAC inhibition , and Brain Derived Neurotrophic Factor (BDNF) signaling