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CD40 — JUN

Text-mined interactions from Literome

Sakata et al., Immunol Lett 1999 : Rapamycin did not inhibit the activation of c-Jun N-terminal kinases (JNKs) induced by anti-CD40 stimulation nor the activation of immediate nuclear transcription factors of NF-kappaB
Afford et al., FASEB J 2001 (Liver Cirrhosis, Biliary) : CD40 activation induced , Fas dependent apoptosis and NF-kappaB/AP-1 signaling in human intrahepatic biliary epithelial cells
Mann et al., J Biol Chem 2002 : CD40 induces interleukin-6 gene transcription in dendritic cells : regulation by TRAF2, AP-1 , NF-kappa B, AND CBF1
Ahmed-Choudhury et al., Mol Biol Cell 2003 : In hepatocytes, CD40 ligation led to sustained up-regulation of AP-1 activity > 24 h associated with increased protein levels of RelA ( p65 ), c-Jun, and c-Fos, whereas no induction of AP-1 activity was observed in IHECs
Lee et al., Mol Cell Biochem 2003 : We previously demonstrated that ROS are generated by cross linking surface receptor CD40 and consequently induce c-Jun N-terminal kinase activation and interleukin-6 secretion in murine B cells
Gallagher et al., Nat Immunol 2007 : Those defects were B cell intrinsic, as MEKK1 was necessary for CD40 mediated activation of the kinases Jnk and p38 and transcription factor c-Jun , as well as for expression of cyclin D2 and activation induced deaminase
Wu et al., Biochem Biophys Res Commun 2009 : LPS stimulation also resulted in phosphorylation of mitogen activated protein kinases, however, only Jun N-terminal kinase (JNK) was partially involved in LPS induced CD40 expression