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CSF3 — IL6

Text-mined interactions from Literome

Hannen et al., Scand J Immunol 1999 : The effect of the IL-6 induced suppression of G-CSF production differed from that of IL-4 and IL-10 in that it was much less pronounced and could be partially overridden by addition of functional IL-1, yet it also appeared to involve the interference with IL-1 function and the suppression of IL-1 independent mechanisms
Carstanjen et al., Transfusion 2002 : G-CSF mobilization treatment induces MMP-9, IL-6 , and soluble VCAM
Akahane et al., Arzneimittelforschung 1990 : The enhanced G-CSF production from NFSA cells may be due at least to interleukin-1 released from muroctasin stimulated macrophages
Leizer et al., Blood 1990 : Cytokine regulation of colony stimulating factor production in cultured human synovial fibroblasts : I. Induction of GM-CSF and G-CSF production by interleukin-1 and tumor necrosis factor
Bharadwaj et al., Cancer Res 2007 (Pancreatic Neoplasms) : IL-6 was able to suppress DC differentiation and G-CSF mainly acted on the suppressing allostimulatory capacity of DCs
Jiang et al., J Int Med Res 2010 (Alzheimer Disease...) : Levels of a7 nAChR protein were significantly increased and levels of interleukin-1ß , tumour necrosis factor-a and nuclear factor-?B ( NF-?B ) protein were significantly decreased in the brain of APP transgenic mice in response to G-CSF
Wang et al., Int J Oncol 1995 : We report that all-trans retinoic acid ( tRA ) synergizes with LPS to enhance the production of granulocyte colony stimulating factor ( G-CSF ) in PMA treated cells, whereas the production of granulocyte-macrophage CSF, interleukin 1-beta (IL-1-beta), and tumor necrosis factor-alpha (TNF-alpha) is minimally affected by tRA
Taga et al., J Exp Med 1987 (Astrocytoma...) : Binding of 125I-BSF-2 to CESS was competitively inhibited by unlabeled BSF-2 but not by IL-1, IL-2, IFN-beta, IFN-gamma, and G-CSF , indicating the presence of the receptors specific for BSF-2
Tanaka et al., Cell 1995 (Listeriosis) : We also demonstrate that NF-IL6 is essential for the induction of G-CSF in macrophages and fibroblasts
Bagby et al., Exp Hematol 1993 (Fanconi Anemia) : In addition, because there was discordance between the G-CSF responses and the GM-CSF and IL-6 responses , the abnormalities noted in IL-1 responsiveness must lie distal to IL-1 receptor function and to stimulus-response coupling pathways shared between the three cytokines
Bataille et al., La Revue du praticien 1993 (Multiple Myeloma) : GM-CSF, IL-3 and G-CSF stimulate the IL-6 responsiveness of myeloma cells without affecting the endogenous IL-6 production
Ogawa et al., Am J Hematol 1996 : IL-2, IL-3, IL-4, IL-5, and IL-6 were unable to induce G-CSF production
Pajkrt et al., Blood 1997 (Endotoxemia) : In group 1, G-CSF significantly enhanced the release of tumor necrosis factor (TNF), interleukin-6 (IL-6), IL-8, IL-1 receptor antagonist (IL-1ra), and soluble TNF receptors ... In group 2, G-CSF significantly reduced IL-8 concentrations and modestly attenuated TNF and IL-6 levels
Weimann et al., J Perinat Med 1998 (Chorioamnionitis...) : G-CSF, GM-CSF and IL-6 levels in cord blood : diminished increase of G-CSF and IL-6 in preterms with perinatal infection compared to term neonates