◀ Back to BCL3

BCL3 — HMOX1

Text-mined interactions from Literome

Tsui et al., Biochem Biophys Res Commun 2005 : Heme oxygenase-1 could induce the expression of anti-apoptotic protein-Bcl-xL in human hepatocyte
Botros et al., Cell Mol Biol Incl Cyto Enzymol 2007 (Hypertension, Renovascular) : In clipped kidneys from 2K1C rats, inhibition of HO activity by SnMP augmented caspase-3 and caspase-9 activity and decreased expression of the anti-apoptotic Bcl-2 protein, while induction of HO-1 with CoPP strongly inhibited the activity of both caspases and increased the induction of Bcl-2 and Bcl-xl proteins
Asija et al., Antioxid Redox Signal 2007 (Hyperglycemia) : Selective expression of HO-1 prevented TNF- and hyperglycemia mediated superoxide ( O2- ) formation, DNA degeneration, and upregulation of caspase, but increased the expression of pAkt and Bcl-xL , proteins responsible for endothelial dysfunction in diabetes
Sue et al., Nephrol Dial Transplant 2009 (Disease Models, Animal...) : We evaluated the protective effects of HO-1 on several apoptotic parameters induced by gentamicin : cleaved caspases-3 and -9, cycloxygenase-2 (Cox-2) and subcellular localization of nuclear factor kappa B-p65 ( NF-kappaB-p65 ), Bcl-xl and HS-1 associated protein ( Hax-1 ) in NRK-52E cells