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PIGR — RELA
Text-mined interactions from Literome
Ackermann et al., Immunology 2004
:
In studies using human intestinal epithelial cells ( HT29 ), multiple inhibitors of the transcription factor
nuclear factor-kappaB (NF-kappaB) , including a dominant negative IkappaBalpha-serine mutant,
inhibited both IL-4- and IFN dependent increases in
pIgR expression
Hinojosa et al., J Infect Dis 2009
(Inflammation...) :
During pneumonia, aged mice had reduced levels of
pIgR and PAFr and less
NFkB activation , despite greater bacterial burden
Bruno et al., Immunol Invest 2010
:
Induction of
pIgR expression in HT-29 cells
required NF-kappaB signaling but not MAPK signaling, in contrast to the requirement for both NF-kappaB and MAPK signaling for induction of pro-inflammatory genes
Bruno et al., Mucosal Immunol 2011
:
Here, we examined the contributions of the
RelA dependent classical and RelB dependent alternative pathways of NF-?B to
pIgR regulation in the HT-29 human IEC line following stimulation with tumor necrosis factor (TNF), lipopolysaccharide ( LPS ; Toll-like receptor 4 (TLR4) ligand ), and polyinosinic : polycytidylic acid ( pIC ; TLR3 ligand )