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ARG2 — JUN

Text-mined interactions from Literome

Christiansen et al., J Immunol 2002 (Rhinitis, Allergic, Perennial...) : We demonstrated that Lys-des-Arg-BK activates the transcription factor AP-1
Peng et al., Blood 2004 : AP1 binding absolutely required Arg218 , Asp222, and Glu225
Liscovsky et al., Immunology 2009 : The use of pharmacological specific inhibitors revealed that arginase activity was dependent on p38 mitogen activated protein kinase ( MAPK ) and extracellular signal regulated protein kinase ( ERK ), but independent of c-Jun N-terminal kinase (JNK) activation
Zhu et al., Am J Physiol Cell Physiol 2010 : Thrombin induces endothelial arginase through AP-1 activation
Yu et al., Clin Cancer Res 2011 (Neoplasm Invasiveness...) : Expression of the FGFR-4 Arg388 protein leads to increased activity of the extracellular signal related kinase ( ERK ) pathway, increased activity of serum response factor (SRF) and AP1 , and transcription of multiple genes that are correlated with aggressive clinical behavior in PCa
Xiong et al., Journal of the American Heart Association 2013 : The activation of p66Shc but not p53 by Arg-II was dependent on extracellular signal regulated kinases ( ERKs ) and sequential activation of 40S ribosomal protein S6 kinase 1 (S6K1)-c-Jun N-terminal kinases (JNKs)