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BCL2 — CRK
Text-mined interactions from Literome
Srivastava et al., Proc Natl Acad Sci U S A 1999
(Breast Neoplasms) :
By contrast, neither an ERK inhibitor ( PD098059 ) nor
p38 inhibitors ( SB203580 and SB202190 )
had an effect on
Bcl-2 phosphorylation
Desbiens et al., Biochem J 2003
:
p38 was also activated by cisplatin only in cells with deregulated expression of c-Myc, but, in contrast with all features of apoptosis, this activation was not
affected by
Bcl-2
Wang et al., J Mol Cell Cardiol 2006
(Inflammation...) :
Following reperfusion, homogenized hearts were analyzed for TNF-alpha, IL-1beta, and IL-6 gene and protein expression,
p38 MAPK
activation , and the apoptosis related proteins caspase-3 and
Bcl-2
Nelyudova et al., Cell cycle (Georgetown, Tex.) 2007
(Cell Transformation, Neoplastic) :
By blocking apoptosis,
Bcl-2 in
p38 dependent manner promotes cell cycle arrest and accelerated senescence after DNA damage and serum withdrawal
Xiang et al., Biochem Biophys Res Commun 2008
:
Furthermore, we found that phenylephrine could significantly upregulate the expression of
Bcl-2 , downregulate the expression of Bax, and
inhibit the activation of both caspase-3 and
p38 mitogen activated protein kinase in autotransplanted SMG
Zhang et al., Biochem Biophys Res Commun 2010
(Anoxia...) :
p38 inhibitor ( SB203580 )
blocked hypoxia induced
Bcl-2 expression, whereas PKC, ERK1/2 and PI3K inhibitors did not