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CA2 — IL3

Text-mined interactions from Literome

Okayama et al., Clin Exp Allergy 1993 (Hypersensitivity, Immediate) : The responsiveness of basophils from atopic donors was variable, only 5/11 subjects showing release of > 10 %, to IL-3 in the range 0.1-100 ng/ml. IL-3 induced histamine release required both extracellular Ca2+ and cell membrane IgE, removal of membrane IgE by lactate stripping or desensitization of basophils by incubation with anti-IgE in a Ca2+-free medium blocking IL-3 induced histamine release
Liu et al., Proc Soc Exp Biol Med 2000 (Calcium Signaling) : Ca2+ release from intracellular stores was strongly stimulated by acetylcholine (ACh) and ATP, but not by norepinephrine ( NA ), epidermal growth factor (EGF), interleukin-6 (IL-6), and tumor necrosis factor-alpha (TNFalpha)
Wang et al., J Biol Chem 2009 (Calcium Signaling) : Protein-tyrosine phosphatase-alpha and Src functionally link focal adhesions to the endoplasmic reticulum to mediate interleukin-1 induced Ca2+ signaling
Kodama et al., Meikai Daigaku Shigaku Zasshi 1990 (Bone Resorption...) : Among several bioactive substances known as coupling factors, transforming growth factor-beta ( TGF-beta ), interleukin-1 (IL-1), and prostaglandin ( PG ) E1 and E2 increased not only the activity of alkaline phosphatase but also the rate of incorporation of 45Ca2+ into ROS 17/2.8 during a 3-day culture : the former two factors are known to be formed at the site where bone is resorbed, while PG 's are known as one of the factors involved in bone resorption
Kodama et al., Meikai Daigaku Shigaku Zasshi 1989 (Bone Resorption) : The effects of interleukin 1, transforming growth factor-beta ( coupling factors ), prostaglandin E1, and prostaglandin E2 on incorporation of 45Ca2+ and on alkaline phosphatase activity were studied using cultured ROS 17/2.8 cells, one of cell lines derived from rat osteosarcoma
Whetton et al., J Cell Sci 1986 : We conclude that IL-3 maintains FDC-Mix 1 cells via its ability to activate PK-C and increase cytosolic levels of Ca2+ , and that an IL-3 mediated activation of PK-C may promote cellular survival via its ability to enhance hexose uptake by phosphorylating the glucose transport protein
Naora et al., J Immunol 1994 : Results of studies performed with use of the Ca2+ ionophore A23187 indicated that elevation of intracellular Ca2+ levels is sufficient to fully induce IL-3 and IL-4 gene expression
Qiu et al., Biochem Pharmacol 1994 : In the present study, we investigated the effects of lymphokines [interleukin-4 (IL-4), interleukin-2 (IL-2) ] and other pharmacologic agents [ lipopolysaccharide (LPS), phorbol 12-myristate 13-acetate ( PMA ) ] on HTMT induced Ca2+ and IP responses in non rosetted cells
MacGlashan et al., J Immunol 1993 : IL-3 also had no effect on resting [Ca2+ ] i levels
Kumar et al., Int J Immunopharmacol 1997 : Effect of prolactin on nitric oxide and interleukin-1 production of murine peritoneal macrophages : role of Ca2+ and protein kinase C