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EGFR — NUP43

Text-mined interactions from Literome

Xie et al., Eur J Biochem 2002 : The signaling pathways that are rapidly elicited by the interaction of ouabain with Na ( + ) /K ( + ) -ATPase, and are independent of changes in intracellular Na ( + ) and K ( + ) concentrations, include activation of Src kinase, transactivation of the epidermal growth factor receptor by Src, activation of Ras and p42/44 mitogen activated protein kinases, and increased generation of reactive oxygen species by mitochondria
Li et al., J Biol Chem 2005 : Tyrosine phosphorylation of Srcasm is dependent on growth factors and the activity of EGFR and SFKs. Increased Srcasm expression enhances p44/42 mitogen activated protein kinase activity and Elk-1 dependent transcriptional events
Nkabyo et al., Am J Physiol Gastrointest Liver Physiol 2005 (MAP Kinase Signaling System) : Inhibitors of EGFR ( AG1478 ) and p44/p42 MAPK ( U0126 ) phosphorylation blocked redox dependent p44/p42 phosphorylation, indicating that signaling occurred by EGFR
Lee et al., American journal of physiology. Renal physiology 2008 : In addition, the PKC inhibitors or an EGFR inhibitor ( AG-1478 ) blocked the BSA induced phosphorylation of p44/42 mitogen activated protein kinases ( MAPKs )