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FOS — RNF19A
Text-mined interactions from Literome
Silvers et al., Neoplasia (New York, N.Y.) 2003
(Skin Neoplasms) :
These results demonstrated that activation of both JNK and
p38 play critical role in UVA mediated
AP-1 transactivation and c-Fos expression in these human keratinocyte cells
Chun et al., Carcinogenesis 2004
:
In order to clarify the
roles of
p38 and ERK in TPA induced
AP-1 activation, we utilized the pharmacologic inhibitors of these enzymes
Gao et al., Zhonghua lao dong wei sheng zhi ye bing za zhi = Zhonghua laodong weisheng zhiyebing zazhi = Chinese journal of industrial hygiene and occupational diseases 2006
:
ERK and JNK, but not
p38 ,
mediated benzo ( a ) pyrene induced cell cycle changes by
AP-1 transactivation in HELF
Tirumurugaan et al., Am J Physiol Lung Cell Mol Physiol 2007
(MAP Kinase Signaling System) :
EMSAs confirmed the
role of
p38 and JNK in mediating NF-kappaB and
AP-1 activation
Humar et al., Int J Biochem Cell Biol 2007
:
Moreover, DNA binding activity of
c-Fos , FosB, and JunB were also
dependent on the
p38 protein kinase activity, whereas JunD, Fra-1 and Fra-2 were not affected
Irarrazabal et al., J Biol Chem 2008
:
Thus,
AP-1 is part of the TonEBP/OREBP enhanceosome, and its role in high NaCl induced activation of TonEBP/OREBP may
require p38 activity
Yang et al., Atherosclerosis 2010
:
N-acetylcysteine, SP600125 ( JNK inhibitor ) and SB203580 (
p38 MAPK inhibitor )
attenuated TNF-alpha induced DNA binding activities of both
AP-1 and NF-kappaB
Son et al., Oncol Rep 2010
(Melanoma, Experimental) :
Ethanol extract of Lycoris radiata induces cell death in B16F10 melanoma via
p38 mediated
AP-1 activation
Butoi et al., Biochim Biophys Acta 2011
:
Monocyte-SMC interaction induced the phosphorylation of
p38MAPK and
activation of
AP-1 transcription factor
Tan et al., J Cell Physiol 2012
:
Moreover, CCN3 induced c-Jun translocation into the nucleus, and the binding of c-Jun to the
AP-1 element on the BMP-4 promoter were both
inhibited by specific inhibitors of the ILK,
p38 , and JNK cascades
Saadane et al., Journal of inflammation (London, England) 2011
:
This was associated with enhanced phosphorylation of
p38 , ERK1/2 and JNK and increased
activation of
AP-1