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ITGB2 — VCAM1

Pathways - manually collected, often from reviews:

• NCI Pathway Database Beta2 integrin cell surface interactions: VCAM1 (VCAM1) → alphaD/beta2 Integrin complex (ITGAD-ITGB2) (modification, collaborate)
  Van der Vieren et al., J Immunol 1999, Kikuchi et al., Cell communication & adhesion 2003*, Grayson et al., J Exp Med 1998*
  Evidence: physical interaction
• NCI Pathway Database Beta2 integrin cell surface interactions: VCAM1 (VCAM1) → alphaD/beta2 Integrin/VCAM1 complex (VCAM1-ITGAD-ITGB2) (modification, collaborate)
  Van der Vieren et al., J Immunol 1999, Kikuchi et al., Cell communication & adhesion 2003*, Grayson et al., J Exp Med 1998*
  Evidence: physical interaction
• NCI Pathway Database Beta2 integrin cell surface interactions: alphaD/beta2 Integrin complex (ITGAD-ITGB2) → alphaD/beta2 Integrin/VCAM1 complex (VCAM1-ITGAD-ITGB2) (modification, collaborate)
  Van der Vieren et al., J Immunol 1999, Kikuchi et al., Cell communication & adhesion 2003*, Grayson et al., J Exp Med 1998*
  Evidence: physical interaction

Text-mined interactions from Literome

Yoshida et al., Aliment Pharmacol Ther 2000 (Helicobacter Infections...) : The expression of CD11b and CD18 on neutrophils and neutrophil dependent adhesion to endothelial cells elicited by HPE were inhibited by lansoprazole and omeprazole at clinical relevant doses, and the expression of ICAM-1 and VCAM-1 on endothelial cells and endothelial dependent neutrophil adherence induced by IL-1beta were also inhibited by lansoprazole and omeprazole at similar doses
Imamoto et al., Biofactors 2004 : The present study revealed that pioglitazone can influence monocyte-EC binding by inhibiting VCAM-1 expression on activated EC and neutrophil-EC binding by inhibiting upregulation of CD11b/CD18 on activated neutrophils