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S100A9 — TP53
Text-mined interactions from Literome
Zilfou et al., Mol Cell Biol 2001
:
Interestingly, unlike retinoblastoma tumor suppressor protein, MDMX, and
p14(ARF) , Sin3
stabilizes p53 in an MDM2 independent manner
Hemmati et al., Oncogene 2002
(Adenocarcinoma...) :
Adenovirus mediated overexpression of
p14(ARF) induces
p53 and Bax independent apoptosis
Javelaud et al., J Biol Chem 2002
(Colonic Neoplasms) :
In accordance with the role of p14(ARF) in p53 stabilization, overexpression of
p14(ARF) in HCT116/p21 ( +/+ ) cells
resulted in a strong increase in
p53 activity
Edwards et al., J Virol 2002
:
Recent data have shown that
p14(ARF) , a positive
regulator of
p53 , inhibits ONYX-015 replication in cells with a wild-type p53, a phenotype that characterizes normal cells
Shamanin et al., Mol Cell Biol 2004
(Cell Transformation, Neoplastic) :
We show that E6Y54D inhibits
p14(ARF) induced activation of
p53 without inactivation of the p53 dependent DNA damage response
Di Ventura et al., PloS one 2008
:
We find that expression of the human p53-Mdm2 module in yeast is sufficient to faithfully recapitulate key aspects of p53 regulation in higher eukaryotes, such as Mdm2 dependent targeting of
p53 for degradation, sumoylation at lysine 386 and further
regulation of this process by
p14(ARF)
Mascaux et al., Eur Respir J 2008
(Bronchial Neoplasms...) :
Murine double minute clone 2 (MDM2),
p14 alternate reading frame (p14arf) , and nucleophosmin (NPM)
regulate p53 activity
Li et al., Biochem J 2009
:
Overexpression of
S100A9 could induce cellular apoptosis, and this was partly
p53 dependent
Khutornenko et al., Proc Natl Acad Sci U S A 2010
:
In epithelial carcinoma cells the activation of
p53 in response to mitochondrial electron transport chain complex III inhibitors does not
require phosphorylation of p53 at Serine 15 or up-regulation of
p14(ARF)
Maruo et al., Proc Natl Acad Sci U S A 2011
:
Moreover, blockade of p16(INK4A) and
p14(ARF) effects on pRb and
p53 by human papilloma virus type 16 E7 and E6 expression, sustained LCL growth after EBNA3C or EBNA3A inactivation