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CSF2 — PRL

Text-mined interactions from Literome

Crispino et al., J Biol Regul Homeost Agents 1992 (Neoplasms) : Effects of granulocyte-macrophage colony stimulating factor on cortisol, growth hormone, prolactin and melatonin in cancer patients ( short communication )
Jacobsen et al., Mol Psychiatry 2012 (Disease Models, Animal...) : Thus, we show that not only low CSF 5-HIAA and a blunted fenfluramine induced prolactin response , but also blunted 5-HT(1A) agonist induced hypothermia and increased 5-HT(2A) receptor levels are bona fide biomarkers of chronic, endogenous 5-HT deficiency
Bates et al., South Med J 1982 (Pseudotumor Cerebri) : This finding suggest an active role for prolactin in CSF production and an etiologic role for prolactin in pseudotumor cerebri
McCann et al., Biol Psychiatry 1994 (Pain) : Outcome measures obtained after at least 2 weeks of drug abstinence included concentrations of monoamine metabolites in cerebrospinal fluid (CSF) , prolactin responses to L-tryptophan, nociceptive responses to ischemic pain, and personality characteristics in which 5-HT has been implicated ( i.e., impulsivity and aggression )
Komorowski et al., Cytobios 1996 : Effect of granulocyte-macrophage colony stimulating factor and granulocyte colony stimulating factor on prolactin and adrenocorticotropic hormone secretion in rats : dose- and time-response in vivo studies ... The in vivo effect of granulocyte-macrophage colony stimulating factor ( GM-CSF ) and granulocyte colony stimulating factor ( G-CSF ) on the plasma levels of prolactin (PRL) and adrenocorticotropic hormone ( ACTH ) in rats were studied
Coccaro et al., Am J Psychiatry 1997 : The Life History of Aggression assessment scores of 24 subjects with personality disorders were compared with their lumbar CSF 5-HIAA concentrations and with their prolactin responses to d-fenfluramine challenge
Sapi et al., J Soc Gynecol Investig 1998 (Breast Neoplasms) : Colony stimulating factor mRNA levels were increased by prolactin and/or insulin in a normal mammary epithelial cell line, while glucocorticoids had no apparent effect on CSF-1 mRNA levels
Coccaro et al., Arch Gen Psychiatry 1998 : We used CSF ( AVP ), CSF 5-hydroxyindoleacetic acid, and the prolactin response to d-fenfluramine challenge ( PRL [ d-FEN ] ) as central indices of vasopressin and serotonergic system function, respectively, in 26 subjects who met the DSM-IV criteria for personality disorder