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FOS — MSH2

Text-mined interactions from Literome

Böhm et al., Ann N Y Acad Sci 1999 : By use of electric mobility shift assays, we demonstrate that alpha-MSH ( 10 ( -6 ) to 10 ( -14 ) M ) activates AP-1 in human dermal fibroblasts, whereas coincubation with interleukin-1 beta (IL-1 beta) results in suppression of its activation
McMinn et al., Am J Physiol Regul Integr Comp Physiol 2000 (Body Weight) : Effect of intracerebroventricular alpha-MSH on food intake, adiposity, c-Fos induction, and neuropeptide expression
Humbert et al., Nucleic Acids Res 2003 : When losing the AP-1 dependent hMSH2 promoter activity, either by mutating the AP-1 binding sites of the hMSH2 promoter or by using a dominant negative c-Jun factor, the hMSH2 overexpression induced by TPA is abolished both in vitro and in vivo
Sabatier et al., J Neurosci 2003 : alpha-MSH induces Fos expression in supraoptic oxytocin neurons, and alpha-MSH melanocortin-4 receptors ( MC4Rs ) are highly expressed in the supraoptic nucleus, suggesting that alpha-MSH and oxytocin actions are not independent ... We confirmed that alpha-MSH induces Fos expression in the supraoptic nucleus when injected centrally and demonstrated that alpha-MSH also stimulates Fos expression in the nucleus when applied locally by retrodialysis ... Thus alpha-MSH induced Fos expression is not associated with electrophysiological excitation of supraoptic neurons because central injection of alpha-MSH or selective MC4 receptor agonists inhibited the electrical activity of oxytocin neurons in the supraoptic nucleus recorded in vivo
Caquineau et al., J Neuroendocrinol 2006 : Similarly, alpha-MSH increased Fos expression in magnocellular oxytocin neurones but had little or no effect in parvocellular oxytocin neurones