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CSF2 — GPI
Text-mined interactions from Literome
Stanford et al., Br J Pharmacol 2000
:
These observations suggest that
PGI ( 2 ), released following the induction of COX-2, differentially
regulates the release of
GM-CSF ( suppresses ) and G-CSF ( potentiates ) from human vascular cells
Lenda et al., J Immunol 2003
(Inflammation...) :
We suggest that during renal inflammation,
CSF-1 mediates M
phi recruitment, proliferation, activation, and, in turn, TEC apoptosis
Takeda et al., Exp Cell Res 2003
:
Granulocyte
colony stimulating factor , which augments neutrophil differentiation of HL-60 cells,
up-regulated GPI-80 expression in the presence of DMSO
Rajotte et al., J Biol Chem 1992
(Leukemia, Megakaryoblastic, Acute) :
Preincubation of the cells with staurosporine, a PKC inhibitor, also significantly reduces the
effect of
GM-CSF or IL-3 on
pHi
Walker et al., Immunobiology 1992
(Fibrosarcoma) :
In the absence of M
phi ,
GM-CSF increased T cell proliferation in response to alloantigen ... The data collectively suggest that TBH M
phi respond differently to GM-CSF, and that tumor induced changes in
GM-CSF responsiveness
affect M phi accessory ability
Bouyer et al., Endocrinology 2007
:
The present study shows that, in the absence of CO ( 2 ) /HCO ( 3 ) ( - ),
CSF-1 causes little change in osteoclast
pH(i)
Kato et al., Cell Immunol 1990
:
We investigated the augmentative
effect of
GM-CSF on M
phi A-cell activity in concanavalin A-stimulated T-cell proliferation ... We observed that
GM-CSF induced M
phi spreading and firm attachment accompanied with enlargement of the cytoplasm, but could not induce de novo expression of Ia antigen ... These results indicate that
GM-CSF augments M
phi A-cell activity through the enhancement of the capacity of M phi to produce IL-1 and IL-6
Hughes et al., Infect Immun 1987
:
Thus,
rGM-CSF does not
induce adequate M
phi activation to kill T. gondii and is not a major microbistatic component of ConAS
Fischer et al., J Immunol 1988
:
The
activation of BMM
phi by
GM-CSF was reversible and could be repeated
Gessl et al., Immunobiology 1995
:
Furthermore,
GM-CSF was able to trigger expression of class II molecules on rat peritoneal macrophages ( M phi ) and BMDM
phi resulted from cultures of BMC with mouse M phi-CSF (M-CSF), and the RT1.B and RT1.D inducing effect of GM-CSF was opposed by M-CSF, and by anti-GM-CSF antibodies
Chen et al., J Immunol 1994
(Cryptococcosis) :
GM-CSF induced AM phi proliferation, but anticryptococcal activity was not due totally to increases in AM phi numbers, indicating AM
phi activation by
GM-CSF
Sasaki et al., Blood 1997
(Hematologic Neoplasms...) :
Phorbol 12-myristate 13-acetate ( PMA ), interleukin-1 (IL-1), IL-3, IL-6, granulocyte-macrophage
colony stimulating factor ( GM-CSF ), thrombopoietin (TPO), and tumor necrosis factor-alpha (TNF-alpha)
enhanced PGI2-R mRNA expression