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ADAMTS4 — TNF

Text-mined interactions from Literome

Bevitt et al., Biochim Biophys Acta 2003 : The expression of ADAMTS genes in ARPE-19 cells and the transcriptional stimulation of some family members by TNFalpha may implicate them in inflammatory eye disease and the compromise of retinal matrix structure, which is evident in age related macular degeneration ( ARMD ) and other retinal pathologies
Liacini et al., Biochem Biophys Res Commun 2005 (Inflammation) : TWHF extract and PG490 also suppressed IL-1-, IL-17-, and TNF-alpha induced expression of ADAMTS-4 in bovine chondrocytes
Cross et al., Brain Res 2006 (Infarction, Middle Cerebral Artery) : In vitro studies suggest that the in vivo modulation of ADAMTS-1 and -4 may be controlled in part by TNF
Wågsäter et al., Atherosclerosis 2008 (Atherosclerosis) : Macrophage expression of ADAMTS-4 , -7, -8 and -9 mRNA were enhanced upon stimulation with IFN-gamma or TNF-alpha
Cecil et al., J Immunol 2009 (Disease Models, Animal...) : However, CD36 transfection in normal human knee immortalized chondrocytes ( CH-8 cells ) was associated with decreased capacity of S100A11 and TNF-alpha to induce chondrocyte hypertrophy and ADAMTS-4 and matrix metalloproteinase 13 expression
Tian et al., Am J Pathol 2013 (Intervertebral Disc Degeneration) : We investigated TNF-a and IL-1ß regulation of ADAMTS-4 expression in nucleus pulposus ( NP ) cells and its role in aggrecan degradation ... Lentiviral transduction with shRNA plasmids shp65, shp52, shIKK-a, and shIKK-ß significantly decreased TNF-a dependent increase in ADAMTS-4 and -5 levels and aggrecan degradation