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JUN — RNF19A

Text-mined interactions from Literome

Oren et al., Mol Cell Biol 1999 : Depolymerization of the cytoskeleton activates JNK and p38 mitogen activated protein kinase and induces c-Jun expression by a signaling pathway that depends on tyrosine kinase activity
Cohen et al., Biochem Pharmacol 2000 (Arteriosclerosis...) : Although other analogues inhibited DNA synthesis, they affected neither ERK1 activation nor p38/stress activated protein kinase 2 or Jun N-terminal kinase 1 activation
Maeda et al., Mol Cell Endocrinol 2003 : SB203580, a specific inhibitor of p38 mitogen activated protein ( MAP ) kinase, and SP600125, a specific inhibitor of c-Jun N-terminal kinase (JNK) , also reduced the induction by both PDGF-BB and bFGF
Silvers et al., Neoplasia (New York, N.Y.) 2003 (Skin Neoplasms) : These results demonstrated that activation of both JNK and p38 play critical role in UVA mediated AP-1 transactivation and c-Fos expression in these human keratinocyte cells
Chun et al., Carcinogenesis 2004 : In order to clarify the roles of p38 and ERK in TPA induced AP-1 activation, we utilized the pharmacologic inhibitors of these enzymes
Welsh et al., Infect Immun 2004 (MAP Kinase Signaling System) : Increases in c-Jun N-terminal kinase/stress activated protein kinase and p38 activity in monocyte derived macrophages following the uptake of Legionella pneumophila
Gao et al., Zhonghua lao dong wei sheng zhi ye bing za zhi = Zhonghua laodong weisheng zhiyebing zazhi = Chinese journal of industrial hygiene and occupational diseases 2006 : ERK and JNK, but not p38 , mediated benzo ( a ) pyrene induced cell cycle changes by AP-1 transactivation in HELF
Tirumurugaan et al., Am J Physiol Lung Cell Mol Physiol 2007 (MAP Kinase Signaling System) : EMSAs confirmed the role of p38 and JNK in mediating NF-kappaB and AP-1 activation
Jiao et al., Zhonghua lao dong wei sheng zhi ye bing za zhi = Zhonghua laodong weisheng zhiyebing zazhi = Chinese journal of industrial hygiene and occupational diseases 2007 : JNK and ERK signaling pathways, but not p38 pathway regulate B ( a ) P-induced c-Jun activation in HELFs
Irarrazabal et al., J Biol Chem 2008 : Thus, AP-1 is part of the TonEBP/OREBP enhanceosome, and its role in high NaCl induced activation of TonEBP/OREBP may require p38 activity
Yang et al., Atherosclerosis 2010 : N-acetylcysteine, SP600125 ( JNK inhibitor ) and SB203580 ( p38 MAPK inhibitor ) attenuated TNF-alpha induced DNA binding activities of both AP-1 and NF-kappaB
Liu et al., Int J Biochem Cell Biol 2010 : Taken together, our data indicate that Fas/FasL up-regulation in piceatannol treated U937 cells is elicited by Ca ( 2+ ) /p38alpha MAPK mediated activation of c-Jun and ATF-2, and suggest that autocrine Fas mediated apoptotic mechanism is involved in piceatannol induced cell death
Son et al., Oncol Rep 2010 (Melanoma, Experimental) : Ethanol extract of Lycoris radiata induces cell death in B16F10 melanoma via p38 mediated AP-1 activation
Ferreiro et al., J Biol Chem 2010 : In addition, p38 recruitment to the osmoinducible gene Cox2 and the TNFa target gene IL8 is mediated by the transcription factors AP1 and NF?B, respectively
Butoi et al., Biochim Biophys Acta 2011 : Monocyte-SMC interaction induced the phosphorylation of p38MAPK and activation of AP-1 transcription factor
Tan et al., J Cell Physiol 2012 : Moreover, CCN3 induced c-Jun translocation into the nucleus, and the binding of c-Jun to the AP-1 element on the BMP-4 promoter were both inhibited by specific inhibitors of the ILK, p38 , and JNK cascades
Saadane et al., Journal of inflammation (London, England) 2011 : This was associated with enhanced phosphorylation of p38 , ERK1/2 and JNK and increased activation of AP-1