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MYLIP — NOS3
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Suárez et al., Circ Res 2007
:
The miRNA expression profile of EC revealed 25 highly expressed miRNAs in human EC and using miRNA mimicry,
miR-222/221 regulates endothelial
nitric oxide synthase protein levels after Dicer silencing
Chan et al., Eur J Pharm Sci 2009
:
We also investigated the
role of
miR-214 on
eNOS expression and in tubulogenesis and motility of HUVEC by transfection of specific miRNA inhibitor or precursor
Perske et al., Am J Pathol 2010
(Carcinoma, Renal Cell...) :
Loss of inducible
nitric oxide synthase expression in the mouse renal cell carcinoma cell line RENCA is
mediated by microRNA
miR-146a
Yamaguchi et al., Atherosclerosis 2011
:
In ES-ECs,
miR-145 overexpression did not induce the synthesis of smooth muscle related proteins nor
suppress the expression of endothelial
nitric oxide synthase
Iaconetti et al., Basic Res Cardiol 2012
(Neointima...) :
Immunoblotting analysis revealed an increased phosphorylation of ERK1/2, JNK/SAPK as well as
eNOS and phospho-eNOS increased expression level in RAO-ECs as a
consequence of
miR-92a inhibition
Boon et al., Thromb Haemost 2012
(Atherosclerosis) :
The down-regulation of
miR-92a by shear stress
enhances the expression of the endothelial
nitric oxide synthase , whereas the up-regulation of miR-19a contributes to the shear stress induced inhibition of cell proliferation
Sun et al., Hypertension 2012
:
Overexpression of miR-155 decreased, whereas inhibition of
miR-155 increased ,
eNOS expression and NO production in human umbilical vein endothelial cells and acetylcholine induced endothelium dependent vasorelaxation in human internal mammary arteries
Zhang et al., Zhonghua Yi Xue Za Zhi 2012
:
The up-regulation of
miR335-3p in old ECs and the up-regulation of miR-135a, miR-96 in the young ECs might
contribute to a lower expression of
eNOS in senescent ECs