Lee et al., Mol Cancer Ther 2006
(Carcinoma, Squamous Cell...) :
We conclude that SOCS-1 methylation status can differentially affect STAT3 activation by IL-6R and EGFR through JAK or MEK in different HNSCC and response to pharmacologic antagonists
Bauer et al., Cell Host Microbe 2012
:
We find that hBD3 is highly active against H. pylori in vitro and is rapidly induced during early infection via EGFR dependent activation of MAP kinase and JAK/STAT signaling