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GPI — MYLIP

Text-mined interactions from Literome

Ahmad et al., Cancer Res 2011 (Breast Neoplasms...) : Moreover, silencing of PGI/AMF expression in MDA-MB-231 cells led to overexpression of miR-200s , which was associated with reversal of EMT phenotype ( i.e., mesenchymal-epithelial transition ), and these findings were consistent with alterations in the relative expression of epithelial ( E-cadherin ) and mesenchymal ( vimentin, ZEB1, ZEB2 ) markers and decreased aggressiveness as judged by clonogenic, motility, and invasion assays
Castagnino et al., PloS one 2013 : PGI ( 2 ) inhibited both Skp2 and miR221/222 expression, but epistasis, ectopic expression, and time course experiments showed that miR-221/222, rather than Skp2, was the primary target of PGI ( 2 ) ... PGI ( 2 ) activates Gs to increase cAMP, and increasing intracellular cAMP phenocopies the effect of PGI ( 2 ) on p27, miR-221/222 , and mitogenesis
Rengaraj et al., Biol Reprod 2013 : In the RNA interference assay, the expression of GPI was greatly regulated by gga-miR-302b and gga-miR-17-5p ... Our results suggested that the regulation of GPI by gga-miR-302b and gga-miR-17-5p affected PGCs proliferation