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Text-mined interactions from Literome

Blix et al., Eur J Oral Sci 1999 (Aggressive Periodontitis) : In this study, the effect of lipopolysaccharides (LPS) extracted from A. actinomycetemcomitans on the expression of the leukocyte adhesion molecules CD11a/CD18, CD11b/CD18, CD11c/CD18 and L-selectin ( CD 62L ) were measured in an ex vivo whole blood system by use of fluorescent antibodies followed by flow cytometry
Roets et al., Am J Vet Res 1999 (Escherichia coli Infections...) : An increase in the number of CD18 receptors on neutrophils is mediated by local production of LPS
Ammon et al., Immunology 2000 : We suggest that the high expression of CD11/CD18 on DC could be involved in the LPS binding of DC
Heinzelmann et al., Immunopharmacology 2000 : On monocytes, LPS and MDP increased surface expression of human leukocyte antigen-DR ( HLA-DR ), CD18 , CD54 ( intercellular adhesion molecule-1, ICAM-1 ), and CD86 ( B7-2 )
Wang et al., Vet Res 2003 : Collectively, results indicate that rbosCD14 inhibit LPS induced increase in CD18 expression and TNF-alpha mRNA
Xiao et al., Yao Xue Xue Bao 2003 : Moreover, LPS induced high expression of ICAM-1 and CD18 was inhibited with treatment of EsA
Collin et al., J Leukoc Biol 2004 (Disease Models, Animal...) : Zileuton also reduced the LPS induced expression of CD11b/CD18 on rat leukocytes
Zhou et al., Am J Physiol Lung Cell Mol Physiol 2005 : LPS activation of Toll-like receptor 4 signals CD11b/CD18 expression in neutrophils
Simms et al., J Surg Res 1992 : In the presence of fibronectin or laminin, LPS reduced CD11b/CD18 expression ( the fibronectin receptor ) as assessed using sheep erythrocytes coated with C3bi
Davtyan et al., Int Immunopharmacol 2006 (Behcet Syndrome) : In BD patients we observed an increase of the spontaneous, N-formyl-Met-Leu-Phe- and LPS induced respiratory burst of monocytes and neutrophils as well as up-regulation of neutrophil CD11a/CD18 surface expression
Couturier et al., J Immunol 1991 : Specific binding of LPS to monocytes was not mediated by the CD11/CD18 complex because mAb to the alpha and beta chains of the Leu-CAM molecules did not alter the binding of LPS to cells and because LPS did not inhibit the binding of labeled mAb to monocytes
Lynn et al., J Immunol 1991 : By using flow cytometry, we have characterized LPS induced expression of the beta 2 integrin CD11b/CD18
Dal-Secco et al., J Immunol 2008 : Neither PAG nor NaHS treatment changed LPS induced CD18 expression on neutrophils, nor did the LPS- and mBSA induced release of neutrophil chemoattractant mediators TNF-alpha, keratinocyte derived chemokine, and LTB ( 4 )
Lynam et al., Blood 1994 : Taken together, the data suggest that inhibition of neutrophil aggregation by LPS/LBP is related to the expression of L-selectin via CD14 rather than LPS inhibition of CD11b/CD18 function during cellular stimulation
Simms et al., Shock 1995 : We investigated the effects of lipopolysaccharide (LPS) on the subcellular location of the integrin receptor CD11b/CD18 ( Mac-1 ) ... Cytoplast and subcellular fractionation experiments were performed to distinguish between receptor shedding and intracellular receptor transport as mechanisms involved for the effects of LPS on CD11b/CD18 expression ... These results demonstrate that LPS regulates CD11b/CD18 expression by inducing intracytoplasmic translocation of this receptor to azurophilic granules
Stewart et al., Surgery 1995 (Acidosis...) : The proportion of circulating neutrophils and lymphocytes was slightly altered 180 minutes after LPS, but there was no obvious effect on T lymphocytes in vivo, and no effect on the LPS induced increase in neutrophil CD18 expression in vitro was seen
Simms et al., J Appl Physiol 1994 (Anoxia) : During hypoxemia ( 10-30 % O2 saturation ), LPS reduced CD11b/CD18 expression ... Incubation of whole blood PMNs with an anti-CD14 monoclonal antibody ( MEM18 ) largely prevented the LPS induced reduction of CD11b/CD18 expression
Paape et al., Am J Vet Res 1996 : To examine Escherichia coli lipopolysaccharide (LPS) effects on expression of CD14 and CD18 cell surface receptors and lectin/carbohydrate mediated nonopsonic phagocytosis of E coli
Kaupke et al., Kidney Int 1996 : G-PDS, as well as the mixtures of spent and fresh G-PDS, significantly depressed the basal PMN expression of adhesion receptors CD11b and CD18 and monocyte expression of CD14, and substantially mitigated the LPS mediated up-regulation of CD11b and CD18
Heinzelmann et al., Cell Immunol 1997 : On monocytes, both LPS and MDP increased the expression of human leukocyte antigen ( HLA ) -DR ( maximal at 6 hr ), CD18 ( maximal at 1-3 hr ), and ICAM-1 ( maximal at 18-24 hr for LPS and 12 hr for MDP ) without increasing the production of superoxide
Nakatomi et al., J Leukoc Biol 1998 : Immobilized LPS induced up-regulation of CD11b/CD18 and latent alkaline phosphatase and also enhanced the adhesive response of neutrophils