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ABL1 — DOK1
Protein-Protein interactions - manually collected from original source literature:
Studies that report less than 10 interactions are marked with *
-
IRef Biogrid Interaction:
DOK1
—
ABL1
(physical association, affinity chromatography technology)
Johnson et al., PloS one 2009*
-
IRef Biogrid Interaction:
DOK1
—
ABL1
(direct interaction, enzymatic study)
Kashige et al., Proc Natl Acad Sci U S A 2000*
-
IRef Biogrid Interaction:
DOK1
—
ABL1
(direct interaction, unspecified method)
Liu et al., Cell communication and signaling : CCS 2012
-
IRef Biogrid Interaction:
DOK1
—
ABL1
(physical association, affinity chromatography technology)
Bhat et al., J Biol Chem 1998*
-
IRef Biogrid Interaction:
DOK1
—
ABL1
(direct interaction, pull down)
Bhat et al., J Biol Chem 1998*
-
IRef Biogrid Interaction:
DOK1
—
ABL1
(direct interaction, enzymatic study)
Yamanashi et al., Cell 1997*
-
IRef Biogrid Interaction:
DOK1
—
ABL1
(physical association, affinity chromatography technology)
Yamanashi et al., Cell 1997*
-
IRef Biogrid Interaction:
DOK1
—
ABL1
(direct interaction, pull down)
van Dijk et al., Blood 2000
-
IRef Hprd Interaction:
DOK1
—
ABL1
(two hybrid)
Cong et al., Mol Cell Biol 1999*
-
IRef Hprd Interaction:
DOK1
—
ABL1
(in vivo)
Cong et al., Mol Cell Biol 1999*
Text-mined interactions from Literome
Dunant et al., Cell Signal 2000
(Leukemia, Myelogenous, Chronic, BCR-ABL Positive) :
Expression of
Bcr-Abl induced tyrosine phosphorylation of both Dok1 and SHIP1 and the formation of a
Dok1/SHIP1 complex
Master et al., J Biol Chem 2003
:
Dok-R binds c-Abl and
regulates Abl kinase activity and mediates cytoskeletal reorganization
Oki et al., Cell cycle (Georgetown, Tex.) 2005
(Cell Transformation, Neoplastic...) :
These data indicate that both SHIP and
Dok1 functionally
regulate the activation of Ras-Erk pathway by
v-Abl and affect the mitogenic activity of v-Abl transformed bone marrow cells