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CA2 — IGF2

Text-mined interactions from Literome

Pecherskaya et al., Alcohol Clin Exp Res 2002 (Alcoholism) : Here, we report that in alcohol exposed myocytes, IGF-I induced augmentation of the cardiac Ca2+ channel activity was absent
Gonzalez et al., J Physiol 2003 : Therefore, IGF-1 prevents age related decline in peak intracellular Ca2+ and specific force in a muscle that does not exhibit changes in fibre type composition with senescence
Kojima et al., Am J Physiol 1992 : In medium containing 10 microM extracellular calcium, IGF-I did not cause any increase in [ Ca2+ ] c. Likewise, blockade of IGF induced calcium entry by either cobalt or tetramethrin abolished IGF-I action on [ Ca2+ ] c. IGF-I mediated oscillation was not affected by either ryanodine or caffeine, compounds that affect calcium induced calcium release
Nishimoto et al., Biochem Biophys Res Commun 1987 : By contrast, IGF-II induces changes in [ Ca2+ ] c in platelet derived growth factor ( PDGF ) - pretreated competent cells : when competent cells are incubated with epidermal growth factor (EGF) for 10 min, subsequent IGF-II induces an immediate increase in [ Ca2+ ] c ... Without EGF treatment, IGF-II does not cause any increase in [ Ca2+ ] c
Mössner et al., J Biol Chem 1984 (Diabetes Mellitus) : Effect of intracellular Ca2+ on insulin-like growth factor II
Martinez et al., J Cell Biochem 1995 : The data in this report show that the osteoblastic Ca2+ response to IGF-II appears to be caused by an intracellular release of Ca2+ which is mediated by the IGF-II/M6P receptor making it possible to envision how the receptor may be an important modulator of osteoblast mediated osteogenesis