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UCSC Genome Browser Gene Interaction Graph
Gene interactions and pathways from curated databases and text-mining

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GCLC — JUN

Protein-Protein interactions - manually collected from original source literature:

Studies that report less than 10 interactions are marked with *

Text-mined interactions from Literome

Manna et al., Oncogene 1999 : Overexpression of gamma-glutamylcysteine synthetase suppresses tumor necrosis factor induced apoptosis and activation of nuclear transcription factor-kappa B and activator protein-1
Urata et al., Free Radic Biol Med 1999 : Melatonin induces gamma-glutamylcysteine synthetase mediated by activator protein-1 in human vascular endothelial cells
Rahman et al., Eur Respir J 2000 (Disease Susceptibility...) : This review describes the redox control and involvement of nuclear factor-kappaB and activator protein-1 in the regulation of cellular glutathione and gamma-glutamylcysteine synthetase under conditions of oxidative stress and inflammation, the role of glutathione in oxidant mediated susceptibility/tolerance, gamma-glutamylcysteine synthetase genetic susceptibility and the potential therapeutic role of glutathione and its precursors in protecting against lung oxidant stress, inflammation and injury
Li et al., J Cell Physiol 2006 : To determine whether age related activation of ERK1/2-AP-1 signaling is responsible for the up-regulation of GCLC , the MEK inhibitors, PD98059 and U0126, were used to block ERK1/2 in VSMC from old rats
Levy et al., Free Radic Biol Med 2009 (Carcinoma, Hepatocellular...) : Despite the increase in binding of phosphorylated c-Jun, reporter assays for EpRE 's showed that inhibition of c-Jun phosphorylation had variable effects on basal and HNE induced transcription of GCLC and GCLM in HBE1 cells
Bergelson et al., Cancer Res 1994 (Carcinoma, Hepatocellular) : We observe that lowering the glutathione levels with buthionine sulfoximine, an inhibitor of gamma-glutamylcysteine synthetase , or diamide, a thiol oxidizing agent, stimulates both basal and chemical-inducible expression of chloramphenicol acetyltransferase activity from EpRE Ya-cat and the AP-1 binding activity