◀ Back to BCL3

BCL3 — EGFR

Text-mined interactions from Literome

Jost et al., J Biol Chem 2001 : Epidermal growth factor receptor dependent control of keratinocyte survival and Bcl-xL expression through a MEK dependent pathway
Bernal et al., Gastroenterology 2006 : Epidermal growth factor receptor signaling regulates Bax and Bcl-w expression and apoptotic responses during intestinal adaptation in mice
Thornburg et al., J Virol 2007 : Induction of epidermal growth factor receptor expression by Epstein-Barr virus latent membrane protein 1 C-terminal activating region 1 is mediated by NF-kappaB p50 homodimer/Bcl-3 complexes ... In this study, the role of NF-kappaB p50 and Bcl-3 in LMP1 mediated upregulation of EGFR was analyzed ... Transient overexpression of p50 and Bcl-3 increased EGFR expression, confirming the regulation of EGFR by these factors ... These data suggest that induction of p50/p50/Bcl-3 complexes by LMP1 CTAR1 mediates LMP1 induced EGFR upregulation and that formation of the p50/p50/Bcl-3 complex is negatively regulated by the p105 precursor
Kung et al., J Virol 2008 : Epstein-Barr virus latent membrane protein 1 induces expression of the epidermal growth factor receptor through effects on Bcl-3 and STAT3 ... The increased nuclear Bcl-3 and p50 homodimer complexes positively regulate EGFR expression
Kung et al., J Virol 2011 : EGFR transcription is induced by LMP1 through a p50 NF?B1-Bcl-3 complex, and Bcl-3 is induced by LMP1 mediated activation of STAT3
Zhao et al., J Lipid Res 2012 : Further studies showed that TNF-a decreased expression of the antiapoptotic proteins Bcl-2 and Bcl-xL , decreased I?Ba and PPAR?, and also inhibited PI3K dependent Akt and EGFR signaling
Rodeck et al., Proc Natl Acad Sci U S A 1997 : Regulation of Bcl-xL expression in human keratinocytes by cell-substratum adhesion and the epidermal growth factor receptor
Stoll et al., Oncogene 1998 : Furthermore, stimulation of EGFR signaling through two natural ligands, transforming growth factor (TGF)-alpha and epidermal growth factor (EGF), increased the expression of Bcl-XL in quiescent keratinocytes and HaCaT cells