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FOS — TRNAE1
Text-mined interactions from Literome
Rourke et al., FEBS Lett 1999
:
As previously noted,
c-Jun/c-Fos heterodimers greatly
increase promoter activity through association with the
CRE/TRE
Cho et al., Infect Immun 2002
(Scrub Typhus...) :
Taken together,
AP-1 activation by both the ERK pathway and the p38 MAPK pathway as well as their binding to
TRE at position -69 to -63 in proximal promoter and TRE at position -2276 to -2270 in enhancer region is altogether essential in induction of MCP-1 mRNA in HUVEC infected with O. tsutsugamushi
Nagashima et al., Mol Cell Endocrinol 2003
(Pituitary Neoplasms) :
Accordingly, PACAP stimulated
AP-1 and CREB transcriptional activity and E ( 2 )
inhibited TRE-LUC but not CRE-LUC activation
de Groot et al., Mol Cell Biol 1991
(Cell Transformation, Viral) :
AP-1 binds to 12-O-tetradecanoylphorbol-13-acetate ( TPA ) -responsive elements ( TREs ) ; therefore, E1A might modulate a specific signal transduction pathway normally induced by activation of the protein kinase C. Binding of jun/AP-1 to a
TRE is
induced in all cell types studied when E1A is expressed
De Cesare et al., Oncogene 1995
:
We report that heterodimerization with
c-Fos does not
increase the binding of c-Jun to the uPA
5'-TRE , in contrast to the increased binding at a consensus AP-1 site