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BCL2 — CRK

Text-mined interactions from Literome

Srivastava et al., Proc Natl Acad Sci U S A 1999 (Breast Neoplasms) : By contrast, neither an ERK inhibitor ( PD098059 ) nor p38 inhibitors ( SB203580 and SB202190 ) had an effect on Bcl-2 phosphorylation
Desbiens et al., Biochem J 2003 : p38 was also activated by cisplatin only in cells with deregulated expression of c-Myc, but, in contrast with all features of apoptosis, this activation was not affected by Bcl-2
Wang et al., J Mol Cell Cardiol 2006 (Inflammation...) : Following reperfusion, homogenized hearts were analyzed for TNF-alpha, IL-1beta, and IL-6 gene and protein expression, p38 MAPK activation , and the apoptosis related proteins caspase-3 and Bcl-2
Nelyudova et al., Cell cycle (Georgetown, Tex.) 2007 (Cell Transformation, Neoplastic) : By blocking apoptosis, Bcl-2 in p38 dependent manner promotes cell cycle arrest and accelerated senescence after DNA damage and serum withdrawal
Xiang et al., Biochem Biophys Res Commun 2008 : Furthermore, we found that phenylephrine could significantly upregulate the expression of Bcl-2 , downregulate the expression of Bax, and inhibit the activation of both caspase-3 and p38 mitogen activated protein kinase in autotransplanted SMG
Zhang et al., Biochem Biophys Res Commun 2010 (Anoxia...) : p38 inhibitor ( SB203580 ) blocked hypoxia induced Bcl-2 expression, whereas PKC, ERK1/2 and PI3K inhibitors did not