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CD69 — IL3

Text-mined interactions from Literome

Lin et al., Biol Neonate 2000 : The effect of interleukin-12 and interleukin-15 on CD69 expression of T-lymphocytes and natural killer cells from umbilical cord blood
Yoshimura et al., J Allergy Clin Immunol 2002 (Asthma) : On the other hand, CD69 expression was only weakly observed in freshly isolated basophils, but IL-3 induced extremely high levels of expression
Zheng et al., Immunology 2002 : IL-3 also up-regulated basophil CD69 surface expression ... The effects of IL-3 on apoptosis and CD69 surface expression of human basophils were completely blocked by LY294002 ( LY ), a potent inhibitor of phosphatidylinositol 3-kinase (PI3-K), but only partially inhibited by lactacystin, a proteasome inhibitor that prevents degradation of IkappaB and NF-kappaB translocation
Yoshimura-Uchiyama et al., Biochem Biophys Res Commun 2003 : However, IL-3 induced CD69 expression preferentially in basophils
Yoshimura-Uchiyama et al., Biochem Biophys Res Commun 2003 : IL-3 was the most potent inducer of CD69 expression after 24-h stimulation, but not after 1-h stimulation
Zhao et al., Leukemia 2008 (Hypersensitivity, Delayed) : sorafenib also caused G ( 0 ) /G ( 1 ) phase arrest, inhibition of CD25 and CD69 expression, interleukin-2 production and LCK phosphorylation in the T cells ; all of these effects exhibited dose and time dependence
Hartnell et al., Immunology 1993 (Asthma) : CD69 expression was also induced by interleukin-3 (IL-3) , IL-5 and interferon-gamma (IFN-gamma), but stimulation of eosinophils with platelet activating factor (PAF) ( 10 ( -6 ) M ) for up to 2 hr did not induce CD69 expression
Wedi et al., J Allergy Clin Immunol 1997 (Asthma...) : In contrast, IL-3 , IL-5, and granulocyte-macrophage-colony stimulating factor caused an upregulated expression of CD69