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CYP19A1 — PRKACG

Text-mined interactions from Literome

Bouchard et al., Endocrinology 2005 (Breast Neoplasms) : PKA mediated phosphorylation increases the interaction between GATA3 and LRH-1 and the requirement for PKA in aromatase PII promoter stimulation involves at least three specific amino acid residues : GATA3 Ser308, GATA4 Ser261, and LRH-1 Ser469
Bulun et al., J Steroid Biochem Mol Biol 2007 (Breast Neoplasms...) : In breast adipose fibroblasts exposed to PGE ( 2 ) secreted by malignant epithelial cells, PKC is also activated, and this potentiates cAMP-PKA dependent induction of aromatase
Chen et al., Cancer Res 2007 (Breast Neoplasms) : Taken together, PGE ( 2 ) activation of p38 and JNK1 via PKA and PKC is necessary for aromatase induction in BAFs, and p38 and JNK1 are potential new drug targets for tissue-specific ablation of aromatase expression in breast cancer