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CDH1 — EPHB2

Text-mined interactions from Literome

Perron et al., Mol Cell Neurosci 1999 : We have found that N-cadherin , as well as laminin ( LN ) and basic fibroblast growth factor (bFGF), can activate ERK in embryonic chick retinal neurons
Krizbai et al., Cell Mol Neurobiol 2005 (MAP Kinase Signaling System) : Furthermore, oxidative stress leads to disruption of the cadherin-beta-catenin complex and an activation of extracellular signal regulated kinase ( ERK1/2 ), which is more intense in the absence of glucose
Bagaeva et al., Tsitologiia 2007 : Study of signal transduction mediated by EGF and HGF in cells with different state of cell adhesion demonstrated that E-cadherin could affect ERK-signal-duration
Wang et al., Mol Biol Cell 2009 (Carcinoma, Non-Small-Cell Lung) : ERK activation also led to induction of metalloproteinase ( MMP)-9 and cleavage of E-cadherin at two specific sites
Zhong et al., Hepatology 2009 (Disease Models, Animal...) : Nevertheless, inhibition of ERK1 resulted in the increased level of E-cadherin in parallel with suppression of TGF-beta1, vimentin, snail, PDGF-BB, BMP4, and p-Smad1
Okumura et al., J Gastroenterol 2010 (Gastrointestinal Neoplasms...) : Inhibition of MEK-ERK signaling mediated up-regulation of E-cadherin and claudin-4, and/or decreased expression of matrix metalloproteinases ( MMPs ) such as MMP-2 and MMP-9, play a role in the TZD induced inhibition of cancer cell invasion
Gupta et al., Int J Oncol 2011 (Cerebellar Neoplasms...) : uPA/ uPAR downregulation also induces E-cadherin expression and inhibits activation of ERK
Ryu et al., Int J Cancer 2012 (Lung Neoplasms...) : Taken together, data indicated that the expression of GSK-3a, ß-catenin and E-cadherin could be negatively regulated by Tß4 induced ERK phosphorylation
Cheng et al., J Clin Endocrinol Metab 2012 : Moreover, the inhibition of EGF induced ERK1/2 , p38, and Akt activation by pharmacological inhibitors attenuated EGF induced Slug expression and the down-regulation of E-cadherin , as well as subsequent cell invasion ... Moreover, the inhibition of EGF induced ERK1/2 , p38, and Akt activation by pharmacological inhibitors attenuated EGF induced Slug expression and the down-regulation of E-cadherin , as well as subsequent cell invasion
Lau et al., PloS one 2013 (Neoplasm Invasiveness...) : The pharmacological inhibition of phosphatidylinositol-3-kinase (PI3K), mammalian target of rapamycin (mTOR), and MEK suggests that both PI3K/Akt/mTOR and MAPK/ERK signaling are required for FGF2 induced E-cadherin down-regulation