◀ Back to TNF

ATP5O — TNF

Text-mined interactions from Literome

Vairo et al., J Cell Physiol 1992 : All three agents stimulated BMM protein synthesis ; TNF alpha and LPS, but not IFN gamma, stimulated BMM Na+/H+ exchange and Na+, K ( + ) -ATPase activities, as well as c-fos mRNA levels
Skayian et al., Life Sci 2006 : Because the Na ( + ) -K ( + ) ATPase is a key player in the contraction of cardiomyocytes, this work was undertaken to study the effect of TNF-alpha on the Na ( + ) -K ( + ) ATPase in rat heart
Kreydiyyeh et al., Prostaglandins Other Lipid Mediat 2007 : The effect of TNF-alpha on liver Na ( + ) -K ( + ) ATPase was studied in Sprague-Dawley rats and in HepG2 cells
Wang et al., J Perinatol 2009 (Pre-Eclampsia) : Effects of Digibind on TNFalpha induced extracellular signal regulated kinase and Na ( + ) /K ( + ) -ATPase expressions were also examined ... The blocking effect was in a concentration dependent manner ; ( 3 ) Digibind had no effects on TNFalpha induced upregulation of extracellular signal regulated kinase phosphorylation, but could block TNFalpha induced downregulation of Na ( + ) /K ( + ) -ATPase beta1 expression
Chan et al., J Food Sci 2009 (Neurodegenerative Diseases) : H ( 2 ) O ( 2 ) or MPP ( + ) treatment significantly decreased Na ( + ) -K ( + ) -ATPase activity, elevated caspase-3 activity and levels of interleukin (IL)-1, IL-6, and tumor necrosis factor (TNF)-alpha ( P < 0.05 ) ; and the pretreatments from these agents significantly restored Na ( + ) -K ( + ) -ATPase activity, suppressed caspase-3 activity and release of IL-1, IL-6, and TNF-alpha ( P < 0.05 )
Bulut et al., Ulus Travma Acil Cerrahi Derg 2011 (Pancreatitis) : While plasma amylase, lipase, IL-1ß, and TNF-a levels, and tissue MDA and MPO levels significantly increased in rats with cerulean induced AP, tissue GSH and Na+-K+-ATPase activity significantly reduced
Dakroub et al., J Cell Biochem 2012 : Sphingosine-1-phosphate is a mediator of TNF-a action on the Na+/K+ ATPase in HepG2 cells ... Treating HepG2 cells with the cytokine in presence of an inhibitor of sphingomyelinase, abrogated the effect of TNF-a on the ATPase
Zhang et al., Basic Res Cardiol 2012 (Calcium Signaling...) : Inhibition of Na/K-ATPase promotes myocardial tumor necrosis factor-alpha protein expression and cardiac dysfunction via calcium/mTOR signaling in endotoxemia ... The aim of this study was to investigate the role of Na/K-ATPase/mTOR signaling in myocardial TNF-a expression during endotoxemia
Pekary et al., J Interferon Cytokine Res 1997 : Tumor necrosis factor-alpha (TNF-alpha) and transforming growth factor-beta 1 ( TGF-beta 1 ) inhibit the expression and activity of Na+/K ( + ) -ATPase in FRTL-5 rat thyroid cells ... Na+/K ( + ) -ATPase activity, which drives iodide uptake by thyroid cells, is inhibited by TNF-alpha and TGF-beta ... The following experiments quantitate the effects of TSH, aging, TNF-alpha , and TGF-beta 1 on the expression and activity of Na+/K ( + ) -ATPase activity in FRTL-5 cells
Teshima et al., Inflamm Res 1998 (Leukemia, Basophilic, Acute) : Ca2+-ATPase inhibitors and PKC activation synergistically stimulate TNF-alpha production in RBL-2H3 cells ... To investigate the effect of Ca2+-ATPase inhibitors on the production of TNF-alpha in rat basophilic leukemia ( RBL-2H3 ) cells ... These results suggest 1 ) that [ Ca2+ ] i increase and subsequent activation of protein kinase C is necessary for the release of TNF-alpha, and they work synergistically, 2 ) that the TNF-alpha release induced by Ca2+-ATPase inhibitors can be regulated at the transcriptional level
Onose et al., Immunol Lett 1998 : The release of TNF-alpha required both a Ca2 ( + ) -ATPase inhibitor and 12-O-tetradecanoylphorbol-13-acetate ( TPA )