◀ Back to MYLIP
MYLIP — VIM
Text-mined interactions from Literome
Park et al., Genes Dev 2008
(Neoplasms...) :
Conversely, inhibition of
miR-200 reduced E-cadherin expression,
increased expression of
Vimentin , and induced EMT
Zhang et al., Clin Exp Metastasis 2009
(Prostatic Neoplasms) :
The lack of
miR-17-3p expression correlated with an
increase in
vimentin synthesis and tumorigenicity
Hur et al., Gut 2013
:
Overexpression of
miR-200c in CRC cell lines caused reduced expression of putative gene targets, and
resulted in increased E-cadherin and reduced
vimentin expression
Shan et al., J Cell Sci 2013
:
In contrast, the passenger strand
miR-17-3p repressed expression of
vimentin , an intermediate filament with the ability to modulate metabolism, and GalNT7, an enzyme that regulates metabolism of liver toxin galactosamine
Li et al., Cancer cell international 2013
:
Silencing of
miR-130b induced E-cadherin expression, while ectopic expression of miR-130b and knockdown of DICER1 increased the expression of
Vimentin , zeb2, N-cadherin, Twist and Snail in EC cells
Arora et al., PloS one 2013
:
miR-506 suppressed the expression of mesenchymal genes such as
Vimentin , Snai2, and CD151 in MDA-MB-231 human breast cancer cell line
Chen et al., Journal of ovarian research 2013
:
However, the stable overexpression of the
miR-200c in the CD117+CD44+CSCs
resulted in a significant down-regulation of ZEB-1 and the
Vimentin expression, an upregulation of the E-cadherin expression as well as a decrease of colony forming, migratory and invasion in vitro